Metastatic rhinosporidioma in a child

Raveenthiran, V

doi:10.1016/j.jpedsurg.2005.12.043

Cited by 13 publications

(14 citation statements)

References 4 publications

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“…1,5 In 1923, Ashworth coined the term Rhinosporidium seeberi after Guillermo Seeber, who submitted a doctoral thesis in 1900. 4 Although rhinosporidiosis has been reported to occur in the Americas, Europe, Africa, and Asia, it is far more common in the tropics, and the greatest prevalence is seen in southern India and Sri Lanka. 1,6 Rhinosporidiosis is transmitted by direct contact with the spores of R seeberi through dust, infected clothing, or fingers, and through swimming in stagnant water contaminated with the spores.…”

Section: Discussionmentioning

confidence: 99%

Laryngotracheal Rhinosporidiosis

Madana

Yolmo²,

Gopalakrishnan

et al. 2013

Ear Nose Throat J

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Rhinosporidiosis is a chronic granulomatous disease caused by Rhinosporidium seeberi. It usually affects mucous membranes of the nose, nasopharynx, and ocular conjunctiva. Cutaneous, laryngeal, tracheal, genital, and bony dissemination is rare. Laryngotracheal involvement poses many diagnostic and therapeutic challenges. A 45-year-old South Indian man presented with complaints of a mass in both nostrils for 2 years, associated with progressive hoarseness of voice and difficulty in breathing for 6 months. Rhinosporidial lesions were seen bilaterally in the nasal cavity. Telescopic and fiberoptic laryngoscopic examinations showed reddish, strawberry-like masses with whitish spots on their surface involving the larynx and trachea. Computed tomography of the head and neck revealed soft-tissue mass lesions involving the bilateral nasal cavities and nasopharynx, extending to the oropharynx and involving the larynx and trachea. A preliminary tracheostomy was performed, followed by direct laryngoscopic excision of the laryngeal lesions and rigid-bronchoscopy-guided excision of the tracheal lesions. The patient was prescribed dapsone and advised to take it for 2 years. At 2 years of follow-up, there was no recurrence.

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Section: Discussionmentioning

confidence: 99%

Laryngotracheal Rhinosporidiosis

Madana

Yolmo²,

Gopalakrishnan

et al. 2013

Ear Nose Throat J

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“…1,5 In 1923, Ashworth named the organism Rhinosporidium seeberi, after Guillermo Seeber, who submitted a doctoral thesis on the topic in 1900. 4 Although rhinosporidiosis has been reported in the Americas, Europe, Africa and Asia, it is far more common in the tropics, with greatest prevalence seen in South India and Sri Lanka. 1,6,7 It may be transmitted by direct contact with the spores of R seeberi via dust or infected clothing, or through swimming in stagnant water contaminated with the spores.…”

Section: Discussionmentioning

confidence: 99%

“…3,10 Medical treatment of rhinosporidiosis with dapsone may be effective, as it arrests the maturation of sporangia, accelerates their degeneration, increases the granulomatous response with fibrosis, and prevents recurrence by interfering with the folic acid metabolism of the organism. 1,4,7,10 …”

Section: Discussionmentioning

confidence: 99%

“…Rhinosporidiosis was first reported in Eastern India in 1894, by O'Kinealy 4 . The disease is caused by Rhinosporidium seeberi , a protist belonging to the mesomycetozoea family of aquatic parasites 1 , 5 .…”

Section: Discussionmentioning

confidence: 99%

“…The disease is caused by Rhinosporidium seeberi , a protist belonging to the mesomycetozoea family of aquatic parasites 1 , 5 . In 1923, Ashworth named the organism Rhinosporidium seeberi , after Guillermo Seeber, who submitted a doctoral thesis on the topic in 1900 4 …”

Section: Discussionmentioning

confidence: 99%

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Rhinosporidiosis of the upper airways and trachea

et al. 2010

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Rhinosporidiosis is a chronic, granulomatous disease which usually affects the mucous membranes of the nose and nasopharynx. Lower dissemination into the trachea is extremely rare. Laryngotracheal involvement poses many diagnostic and therapeutic challenges, due to the potential risk of bleeding and aspiration. In the presented case, local spread of rhinosporidiosis, due to direct spillage of spores from the nasopharynx into the larynx during episodes of bleeding or previous surgery, may have resulted in laryngotracheal involvement, although systemic spread cannot be excluded.

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