Migraine Prevalence: A Literature Review

Sheffield, Reinée E.

doi:10.1046/j.1526-4610.1998.3808595.x

Cited by 47 publications

(34 citation statements)

References 13 publications

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“…The prevalence of migraine in the world is about 10% and it is more prevalent in women [1]. The pathogenesis of migraine is still unclear, but evidences suggest the role of inflammation and vascular dysfunction in pain generation.…”

Section: Introductionmentioning

confidence: 99%

Association of the long pentraxin PTX3 gene polymorphism (rs3816527) with migraine in an Iranian population

Zandifar

Iraji

Taheriun

et al. 2015

Journal of the Neurological Sciences

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Section: Introductionmentioning

confidence: 99%

Association of the long pentraxin PTX3 gene polymorphism (rs3816527) with migraine in an Iranian population

Zandifar

Iraji

Taheriun

et al. 2015

Journal of the Neurological Sciences

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“…It is characterized by recurrent attacks of typically severe, pulsating and unilateral headache, and associated symptoms such as nausea, vomiting, photo-and phonophobia [8]. The estimated worldwide prevalence of migraine is about 10% [25] with a two to threefold higher prevalence in females (15-18%) than in males (6%) [26]. Migraine attacks are classified into two subtypes: migraine with aura (MA) and migraine without aura (MO) [15].…”

Section: Introductionmentioning

confidence: 99%

Investigation of TNFA 308G > A and TNFB 252G > A polymorphisms in genetic susceptibility to migraine

et al. 2009

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We aimed to look for the association of tumor necrosis factor (TNF) gene polymorphisms (TNFA 308G > A, and TNFB 252G > A) in genetic susceptibility to migraine. The pathogenesis of migraine involves many immune-mediated mechanisms in the vascular endothelium. TNF, being a potent immunomodulator and pro-inflammatory cytokine, is suggested to be involved in inflammatory reactions leading to migraine attacks. A total of 216 normotensive migraine patients, 160 tension type headache (TTH) patients and 216 healthy controls (HC) were recruited in the study. The genetic polymorphisms were investigated through SNP association analysis using a matched case control migraine population. Genotyping of TNFA 308G > A polymorphism and TNFB 252G > A was done using ARMS PCR and PCR-RFLP, respectively. A borderline association was observed in TNFA 308GA genotype in migraine patients versus HC (p = 0.043; OR = 1.763; 95% CI = 1.019-3.051). After sub-grouping migraine into migraine with aura (MA) or without aura, significant difference at genotypic (p = 0.015; OR = 2.293; 95% CI = 1.172-4.487) as well as allelic (p = 0.035; OR = 1.955; 95% CI = 1.047-3.651) level was evident. The difference was even more significant in female MA at genotypic (p = 0.006; OR = 2.901; 95% CI = 1.361-6.181) and allelic level (p = 0.017; OR = 2.318; 95% CI = 1.159-4.635) as well as for A allele carriers in MA [p value = 0.020; OR = 2.205 (1.132-4.295)] and female MA (p value = 0.008; OR = 2.741; CI = 1.297-5.792). No association of TNFB252G > A was observed in migraine patients or any subgroups. We did not find any association of TNFA or TNFB gene polymorphisms with TTH. In conclusion, the TNFA 308G > A polymorphism was found to be associated with MA, particularly in females, whereas we could not find any association of TNFB 252G > A polymorphism in genetic susceptibility to migraine on comparing the migraine patients with HC or TTH patients.

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“…3 For both men and women, the prevalence of migraine rises throughout early adult life and falls after midlife. In girls and women, the rate almost triples between age 10 and 30 years.…”

Section: Epidemiologymentioning

confidence: 99%

Foods and Supplements in the Management of Migraine Headaches

Sun-Edelstein¹,

Mauskop²

2009

The Clinical Journal of Pain

114

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Objective: Although a wide range of acute and preventative medications are now available for the treatment of migraine headaches, many patients will not have a significant improvement in the frequency and severity of their headaches unless lifestyle modifications are made. Also, given the myriad side effects of traditional prescription medications, there is an increasing demand for ''natural'' treatment like vitamins and supplements for common ailments such as headaches. Here, we discuss the role of food triggers in the management of migraines, and review the evidence for supplements in migraine treatment.Methods: A review of the English language literature on preclinical and clinical studies of any type on food triggers, vitamins, supplements, and migraine headaches was conducted.Results: A detailed nutritional history is helpful in identifying food triggers. Although the data surrounding the role of certain foods and substances in triggering headaches is controversial, certain subsets of patients may be sensitive to phenylethylamine, tyramine, aspartame, monosodium glutamate, nitrates, nitrites, alcohol, and caffeine. The available evidence for the efficacy of certain vitamins and supplements in preventing migraines supports the use of these agents in the migraine treatment. Conclusions:The identification of food triggers, with the help of food diaries, is an inexpensive way to reduce migraine headaches. We also recommend the use of the following supplements in the preventative treatment of migraines, in decreasing order of preference: magnesium, Petasites hybridus, feverfew, coenzyme Q10, riboflavin, and alpha lipoic acid.

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Migraine Prevalence: A Literature Review

Cited by 47 publications

References 13 publications

Association of the long pentraxin PTX3 gene polymorphism (rs3816527) with migraine in an Iranian population

Association of the long pentraxin PTX3 gene polymorphism (rs3816527) with migraine in an Iranian population

Investigation of TNFA 308G > A and TNFB 252G > A polymorphisms in genetic susceptibility to migraine

Foods and Supplements in the Management of Migraine Headaches

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