2006
DOI: 10.4321/s0212-71992006000200015
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Miositis viral aguda del adulto

Abstract: denciado un riesgo de recurrencia del 50-78% tras suspenderla. Por este motivo, algunos autores recomiendan su mantenimiento a largo plazo (5,10). Tanto en nuestro caso, como en los anteriormente citados, los niveles de anticuerpos se normalizaron tras iniciar el tratamiento anticoagulante. En algunos casos, se sugiere la utilización de antibióticos de forma coadyuvante ya que uno de los posibles desencadenantes son los agentes infecciosos (8).

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“…Intense physical exercise, trauma, generalized tonic-clonic seizures, acute psychosis, systemic connective tissue diseases, renal or cardiac failure, viral diseases that cause myopathies such as influenza, coxsackie, adenovirus, prostate cancer, celiac disease, obstructive sleep apnea, moderate to severe hypothyroidism, drugs such as statins, fibrates, antiretrovirals, beta-blockers, clozapine, angiotensin II receptor antagonists, hydroxychloroquine, isotretinoin, and colchicine; neuromuscular diseases such as Guillain Barré, amyotrophic lateral sclerosis, myopathy due to the use of corticosteroids, hyperthyroidism, collagen diseases, alcoholism, procedures such as intramuscular injections and electromyography have been described as possible etiologies. [6][7][8][9][10][11][12] Besides that, the most frequent causes of CPK elevation are genetic diseases such as muscular dystrophies, congenital myopathies, channelopathies, mitochondrial myopathies, and myotonic dystrophies, although exercise can also temporarily increase CPK levels due to rhabdomyolysis. 2,13 Muscular dystrophies are classified according to their genetic transmission mechanism, either autosomal dominant or recessive, and those linked to the X chromosome.…”
Section: Introductionmentioning
confidence: 99%
“…Intense physical exercise, trauma, generalized tonic-clonic seizures, acute psychosis, systemic connective tissue diseases, renal or cardiac failure, viral diseases that cause myopathies such as influenza, coxsackie, adenovirus, prostate cancer, celiac disease, obstructive sleep apnea, moderate to severe hypothyroidism, drugs such as statins, fibrates, antiretrovirals, beta-blockers, clozapine, angiotensin II receptor antagonists, hydroxychloroquine, isotretinoin, and colchicine; neuromuscular diseases such as Guillain Barré, amyotrophic lateral sclerosis, myopathy due to the use of corticosteroids, hyperthyroidism, collagen diseases, alcoholism, procedures such as intramuscular injections and electromyography have been described as possible etiologies. [6][7][8][9][10][11][12] Besides that, the most frequent causes of CPK elevation are genetic diseases such as muscular dystrophies, congenital myopathies, channelopathies, mitochondrial myopathies, and myotonic dystrophies, although exercise can also temporarily increase CPK levels due to rhabdomyolysis. 2,13 Muscular dystrophies are classified according to their genetic transmission mechanism, either autosomal dominant or recessive, and those linked to the X chromosome.…”
Section: Introductionmentioning
confidence: 99%