2022
DOI: 10.21203/rs.3.rs-2151273/v1
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miRNA-485 Regulates Th17 Generation and Pathogenesis in Experimental Autoimmune Encephalomyelitis through Targeting STAT3

Abstract: Experimental autoimmune encephalomyelitis (EAE) refers to the T-helper (Th) cell-induced autoimmune disease causing demyelination, axonal loss, as well as neurodegeneration of central nervous system (CNS). EAE pathogenesis is highly dependent on T-helper 17 cells (Th17) that generate interleukin-17 (IL-17), and their activity and differentiation are tightly regulated by some cytokines and transcription factors (TFs). In the pathogenic mechanism of various autoimmune disorders, including EAE, certain miRNAs pla… Show more

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