The coronoid process is an anatomical part of the mandible that serves as the attachment for the temporalis muscle, buccinator muscle, and the anterior part of the masseter muscle. In classic anatomy, it is described as a sharp triangular-shaped structure in extension of the anterior border of the mandibular ramus. In reality, this structure shows great morphological variety, including hook-shaped type, triangular type, or rounded type. In the study of Lalitha and Sridevi 1 , the majority (73.9%) of mandibles had the same type of coronoid process on both sides, while 26.1% of the cases showed different types on the two sides. Allometric variation can be established in mandibular shape in humans, with taller individuals having superoinferiorly taller rami with more anteriorly-oriented and higher coronoid processes and a corresponding deeper sigmoid notch 2 .Mandibular coronoid process hyperplasia (MCPH) is an uncommon congenital or developmental condition that is characterized by a macroscopic increase in the size of the coronoid process with a normal histologic structure of the bone. MCPH can exist as a uni-or bilateral condition and causes a slow and progressive reduction of mouth opening. Restricted mouth opening results from impingement of the coronoid process on the medial surface of the zygomatic arch [3][4][5] . Unilateral MCPH can involve facial asymmetry with deviation toward the affected side 5 . Langenbeck was the first to report MCPH in 1853, and the first case of restricted mouth opening due to coronoid process enlargement was reported in 1899 by Jacob 3 . Jacob's disease refers to the condition where the coronoid process creates a new joint with the zygomatic process.The pathogenesis of MCPH remains unclear. Several factors might be associated with development of MCPH 6,7 . History of facial trauma and, in particular, zygomatic arch trauma is thought to be a contributing factor in some cases [8][9][10][11]