1980
DOI: 10.1016/s0022-5347(17)56131-5
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Neonatal Testicular Infarction and Polycythemia

Abstract: Torsion has been observed in only 50 per cent of the reported cases of neonatal testicular infarction. We report a neonate in whom polycythemia and unilateral testicular infarction were present at birth. Although cause and effect are discussed additional reports are needed to confirm whether in utero testicular infarction was the result of in utero polycythemia.

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Cited by 4 publications
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“…Interrup tion of the venous drainage produces a marked edema, congestion, rupture of the venous wall, and interstitial hemorrhage. This occurs in the torsion of the spermatic cord [12,13], Since this torsion is not always demonstra ble [14], other causes, such as polycythemia and hyper viscosity in the newborn [15], venous compression by either hydrocele or an incarcerated hernia [16][17][18], and venal caval thrombosis [19] have been reported.…”
Section: Discussionmentioning
confidence: 99%
“…Interrup tion of the venous drainage produces a marked edema, congestion, rupture of the venous wall, and interstitial hemorrhage. This occurs in the torsion of the spermatic cord [12,13], Since this torsion is not always demonstra ble [14], other causes, such as polycythemia and hyper viscosity in the newborn [15], venous compression by either hydrocele or an incarcerated hernia [16][17][18], and venal caval thrombosis [19] have been reported.…”
Section: Discussionmentioning
confidence: 99%