Neutron Cross Sections for Cadmium Isotopes

EPR-Proton Qubits’ Role in Evolution and Age-Related Disease

“…Under these conditions, interactions between superpositions of entangled states [7][8][9][10][11] and water, ions and/or random temperature fluctuations [12] would cause rapid decoherence [13,14], which would disallow quantum contributions to normally observed biological reactions. Nevertheless, operational molecular genetic systems exist [15][16][17][18][19][20][21][22][23][24][25][26] that routinely exhibit "normal" molecular genetic reactions which are incompatible with classical Watson-Crick [27,28], but are internally consistent with EPRgenerated [29][30][31][32][33][34] entangled proton qubits [35][36][37][38][39], subjected to measurements by Grover's-type [40] quantum processors [41][42][43][44][45]. Dynamic quantum information processing [15][16][17] examples exhibited by ancient [46] T4 phage DNA [47]…”

“…Based on the present and previousassessments [35][36][37][38][39], quantum information processing [40][41][42][43][44][45] exhibited by prokaryotic [15][16][17]20,21] and eukaryotic [47][48][49][50][51][52][53][54] genomic systems can be evolutionarily explained in terms of EPR-generated [29][30][31] entangled proton qubits originating in primordial RNA -ribozyme duplex segments [35,36,[57][58][59][60][61][62][63], which subsequently were quantum mechanically processed by Grover's-type [40] quantum readers. The molecular genetics history of observing [18][19][20][21][22][23][24][25], but misidentifying, time-dependent EPR-generated [29][30]…”

“…The molecular genetics history of observing [18][19][20][21][22][23][24][25], but misidentifying, time-dependent EPR-generated [29][30][31]-keto-amino -(entanglement) → enol-imine -entangled proton qubits ( Figure 1-4) has enabled perpetration of a falsifiable molecular genetics model, i.e., the classical molecular clock [15][16][17]27,28]. In these cases [18][19][20][21][22][35][36][37][38][39][49][50][51]54], random classical processes [12] subjected metabolically inert [64], metastable hydrogen bonded amino (−NH2) DNA proton systems [65] to quantum uncertainty limits [2,66], Δx Δpx ≥ ħ/2. This introduced a probability of direct quantum mechanical proton -proton interaction, yielding EPRarrangements [29][30][31], keto-amino -(entanglement) → enol−imine, observed [15][16][17] as G-C → G´-C´, G-C → *G-*C and A-T → *A-*T. (G´-C´, *G-*C,*A-*T-denote necessity of Hilbert space to describe dynamics of embedded entangled proton qubits; see Figure 2…”

“…In these cases, the wild-type r + allele requires expression (transcription and translation) yielded by classical biological operations on physical substitutions, i.e., G → T and/or C → T, before DNA growth can occur on E. coli K [23,73]. Curiously, heteroduplex heterozygote T4 phage ts systems [15][16][17]23] -i.e., time-dependent "point" mutations, G-C → G´-C´, G-C → *G-*C, A-T → *A-*T, that accumulate in metabolically inert suspensions of T4 Phage particles [18][19][20][21][22][23] -routinely exhibit identical G′ → T and *C → T mutation frequencies expressed by prereplication transcription, and post-transcription replication [16,17,20,21]. (Symmetric, asymmetric and second asymmetric (unlabeled) channels (→) by which metastable keto-amino G-C protons populate enol and imine entangled proton qubit states.…”

“…Consequently, classical experimental molecular genetic investigations to provide a rationale for nonclassical, Gʹ → T &*C → T transcription and replication observations [15][16][17][18][19][20][21][22] were ultimately abandoned [27]. The nonclassical, identical G′ → T and *C → T mutation frequencies, expressed by pre-replication transcription and post-transcription replication [20,21], are also exhibited by evolving microsatellite [52,74], short tandem repeats(STRs) within human and rat genomes [35,38].…”