Non-cardiogenic pulmonary oedema due to foreign body aspiration

Pahade, Avinash; Green, K. M. J.; Carpentier, J.-P.

doi:10.1017/s0022215100158074

Cited by 6 publications

(5 citation statements)

References 8 publications

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“…Whether the addition of diuretics plays a role in the resolution of POPE is not clear. 2,3 Mild pulmonary edema can be treated with supplemental oxygen alone. More severe pulmonary edema may require intubation and positive pressure ventilation.…”

Section: Discussionmentioning

confidence: 99%

“…Post-obstructive pulmonary edema (POPE) has most frequently been described after an upper airway obstruction. [1][2][3] We report the case of a 13-month-old boy who developed POPE following acute respiratory distress from an obstruction of the distal trachea following aspiration of a foreign body (FB). This case illustrates the importance of considering FB aspiration as a cause of POPE.…”

Section: Introductionmentioning

confidence: 99%

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Post-obstructive pulmonary edema from aspirated nuts

Bashir

Ahmad

Silverman

et al. 2017

SAGE Open Medical Case Reports

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Objectives:Post-obstructive pulmonary edema is thought to occur from hemodynamic changes secondary to forced inspiration against the closed airway due to acute or chronic airway obstruction. We report a case of a 13 month-old boy who developed pulmonary edema from aspirated foreign body, nuts.Methods:He underwent emergency bronchoscopy to confirm the clinical diagnosis of aspirated nuts in the trachea and nuts were removed endoscopically. His trachea was then intubated and he was mechanically ventilated with oxygen.Results:He developed florid pulmonary edema early in the course with tracheal obstruction and during endoscopic removal of nuts. After removal of obstruction he was ventilated mechanically and pulmonary edema cleared rapidly.Conclusions:Aspirated nuts obstructing trachea can induce obstructive pulmonary edema. Early recognition of foreign body obstruction based on clinical history and its removal resolved pulmonary edema.

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Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Post-obstructive pulmonary edema from aspirated nuts

Bashir

Ahmad

Silverman

et al. 2017

SAGE Open Medical Case Reports

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“…The negative intrathoracic pressure may also impede left‐sided cardiac function via decreasing left ventricular filling and increasing left ventricular afterload, both of which in turn can result in increased pulmonary capillary pressure as well 85 . Additionally, some researchers suggest that the dyspnea associated with airway obstruction causes an increased sympathetic surge similar to that seen in NPE 7,88,90 …”

Section: Postobstructive Pulmonary Edemamentioning

confidence: 97%

“…POPE occurs in people secondary to laryngospasm, foreign body inhalation, croup, epiglottitis, strangulation, and goiter. 7,[87][88][89] The proposed pathophysiologic mechanism by which upper airway obstruction can lead to pulmonary edema is based on forcible inspiration against a closed epiglottis, which decreases intrathoracic pressure dramatically. In young healthy adults, these pressures can get as low as -140 cm H 2 O.…”

Section: Pathophysiologymentioning

confidence: 99%

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Noncardiogenic pulmonary edema in small animals

Unger

Martin

2023

J Vet Emergen Crit Care

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Objective To review various types of noncardiogenic pulmonary edema (NCPE) in cats and dogs. Etiology NCPE is an abnormal fluid accumulation in the lung interstitium or alveoli that is not caused by cardiogenic causes or fluid overload. It can be due to changes in vascular permeability, hydrostatic pressure in the pulmonary vasculature, or a combination thereof. Possible causes include inflammatory states within the lung or in remote tissues (acute respiratory distress syndrome [ARDS]), airway obstruction (post‐obstructive pulmonary edema), neurologic disease such as head trauma or seizures (neurogenic pulmonary edema), electrocution, after re‐expansion of a collapsed lung or after drowning. Diagnosis Diagnosis of NCPE is generally based on history, physical examination, and diagnostic imaging. Radiographic findings suggestive of NCPE are interstitial to alveolar pulmonary opacities in the absence of signs of left‐sided congestive heart failure or fluid overload such as cardiomegaly or congested pulmonary veins. Computed tomography and edema fluid analysis may aid in the diagnosis, while some forms of NCPE require additional findings to reach a diagnosis. Therapy The goal of therapy for all types of NCPE is to preserve tissue oxygenation and reduce the work of breathing. This may be achieved by removing the inciting cause (eg, airway obstruction) and cage rest in mild cases and supplemental oxygen in moderate cases and may require mechanical ventilation in severe cases. Prognosis Prognosis is generally good for most causes of veterinary NCPE except for ARDS, although data are scarce for some etiologies of NCPE.

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Anesthésie pour extraction d’un corps étranger trachéobronchique

Baujard

2012

Le Praticien en Anesthésie Réanimation

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Non-cardiogenic pulmonary oedema due to foreign body aspiration

Cited by 6 publications

References 8 publications

Post-obstructive pulmonary edema from aspirated nuts

Post-obstructive pulmonary edema from aspirated nuts

Noncardiogenic pulmonary edema in small animals

Anesthésie pour extraction d’un corps étranger trachéobronchique

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