Nonconvulsive Status Epilepticus as a Cause for Delayed Emergence after Electroconvulsive Therapy

Crider, Bruce A.; Hansen-Grant, Sherri

doi:10.1097/00000542-199502000-00033

Cited by 19 publications

(6 citation statements)

References 15 publications

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“…Prolonged somasthetic or visceral sensory disturbances may represent sensory status (28). NCSE may produce diminished responsiveness following electroconvulsive therapy (29).…”

Section: Presentationmentioning

confidence: 99%

Presentation, Evaluation, and Treatment of Nonconvulsive Status Epilepticus

Drislane¹

2000

Epilepsy & Behavior

151

109

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“…Prolonged somasthetic or visceral sensory disturbances may represent sensory status (28). NCSE may produce diminished responsiveness following electroconvulsive therapy (29).…”

Section: Presentationmentioning

confidence: 99%

Presentation, Evaluation, and Treatment of Nonconvulsive Status Epilepticus

Drislane¹

2000

Epilepsy & Behavior

151

109

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“…However, ECT has been successfully repeated after tardive seizures without further complications. In these limited case reports, there appears to have been an offending agent potentially lowering the seizure threshold such as a beta lactam 6 or theophylline 8 . After its removal, ECT treatments were not associated with further tardive seizures.…”

Section: Discussionmentioning

confidence: 99%

Tardive Seizure With Postictal Aphasia

Felkel¹,

Wagner²,

Kimball³

et al. 2012

The Journal of ECT

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Electroconvulsive therapy (ECT) is a highly effective treatment for certain psychiatric disorders with relatively few serious side effects or complications. Tardive seizures are one of these rare, but potentially fatal complications. Recognizing and treating tardive seizures is essential to prevent prolonged postictal confusion, progression to status epilepticus and associated soft tissue injury, anoxia, aspiration and death. Currently there is an unknown prevalence of their occurrence and an overall lack of clinical description of their phenomenology. We describe a case in which a patient develops a tardive seizure followed by a receptive and expressive aphasia, thought to be a variant of Todd’s postictal paralysis. This case is further unique in that there was a lateralization of a motor seizure presumably to the hemisphere contralateral to the RUL electrode placement.

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“…13 Status epilepticus in the form of both convulsions and non-convulsive seizures following ECT has been reported in the literature but is a rare occurrence. [14][15][16][17][18][19] Associated factors include hypoxia, lowered seizure threshold due to medications or pre-existing epileptiform activity. 14,15 Pregnancy may alter the seizure threshold in unpredictable ways.…”

Section: Discussionmentioning

confidence: 99%

“…[14][15][16][17][18][19] Associated factors include hypoxia, lowered seizure threshold due to medications or pre-existing epileptiform activity. 14,15 Pregnancy may alter the seizure threshold in unpredictable ways. During pregnancy, hormonal influences, as well as other factors such as sleep deprivation, electrolyte disturbances, fatigue and stress, may lower seizure threshold and affect ECT outcome.…”

Section: Discussionmentioning

confidence: 99%

“…5 Status epilepticus can occur more commonly with the multiple monitored technique, in which several seizures are induced during one period of anesthesia; however, it can also occur with bilateral and unilateral single treatments. 14,18,19 Although both the safety and efficacy of ECT during pregnancy are addressed in the literature, it is important to be prepared for both maternal complications and the fetal consequences. The need for high doses of anesthetic agents to suppress seizure activity led to further complications in our patient in the form of hypotension, diabetes insipidus, and cardiac and renal dysfunction.…”

Section: Discussionmentioning

confidence: 99%

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