Noninvasive Multimodality Imaging for the Diagnosis of Constrictive Pericarditis

Alajaji, Wissam; Xu, Bo; Sripariwuth, Apichaya; Menon, Vivek; Kumar, Arnav; Schleicher, Mary; Isma’eel, Hussain; Cremer, Paul; Bolen, Michael A.; Klein, Allan L.

doi:10.1161/circimaging.118.007878

Cited by 55 publications

(64 citation statements)

References 62 publications

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“…It also determines the extent of the pericardial inflammation and provides input to guide treatment [ 5 ]. CT scan can reveal the presence of pericardial calcification in 25% of patients, and aid in the pre-operative assessment [ 6 ].…”

Section: Discussionmentioning

confidence: 99%

Constrictive Pericarditis: An Unusual Presentation of Rheumatoid Vasculitis

Kharsa¹,

Chowdhury²,

Tan³

et al. 2022

Cureus

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Section: Discussionmentioning

confidence: 99%

Constrictive Pericarditis: An Unusual Presentation of Rheumatoid Vasculitis

Kharsa¹,

Chowdhury²,

Tan³

et al. 2022

Cureus

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“…Myocarditis is an inflammatory disease with numerous causes and complicated pathophysiological mechanism, CMR can provide valuable information for treatment of myocarditis both for clinical care and future research, similar to the situation with pericarditis (6)(7)(8). Future clinical trials, comparing treatment guided with and without imaging will be necessary.…”

Section: Discussionmentioning

confidence: 99%

Discordant clinical presentation and pathophysiology: insights with cardiac magnetic resonance

Zhao

Wang

et al. 2019

Cardiovasc. Diagn. Ther

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Case presentationA 23-year-old man was admitted to our hospital with hemodynamically stable wide-complex ventricular tachycardia (VT) for electrophysiological examination and potential ablation therapy. Three years prior to this admission, the patient had presented with palpitation and loss of consciousness while playing basketball. An electrocardiogram showed wide-complex VT and Holter monitoring identified 4,814 premature ventricular beat/24 h with 2 different morphologies. However, electrophysiological examination failed to induce the ventricular arrhythmias. The patient refused implantation of an implantable cardiac defibrillator and was discharged on metoprolol. Two years ago, the patient presented with chest distress and fatigue. He was diagnosed with "myocarditis" and received supportive treatment in other hospital. He continued to have episodes of chest pain and 1 week prior to admission he presented with chest pain, palpitation and syncope. He was then referred to our tertiary center. Detailed review of available but limited medical records did not provide evidence to support or ruleout inflammatory injury of the myocardium prior to or at the time of the initial presentation with VT. Further documentation about the prior diagnosis and treatment of myocarditis was insufficient; neither imaging studies nor endomyocardial biopsy (EMB) were performed.During the current admission, a chest X-ray was unremarkable. Initial labs were significant for elevated thyroid peroxidase antibody (TPOAb) of 779.7 IU/mL (reference range, 0-9 IU/mL), and thyroglobulin antibody (TgAb) of 15.1 IU/mL (reference range, 0-4.9 IU/mL), but total triiodothyronine (TT3), total thyroxine (TT4), human thyroid-stimulating hormone (hTSH), free-T3 (FT3), free-T4 (FT4) were normal, troponin-I (TNI), creatine-kinase MB (CKMB), myoglobin (MYO) and whole blood count (WBC) were in the normal range (Table 1). Ultrasound exam showed heterogeneity of thyroid parenchyma. EMB was recommended, but the patient refused. Cardiac magnetic resonance (CMR) imaging was

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“…Although both conditions shares E-wave predominance and short deceleration time, respirophasic shifting of the interventricular septum, caused by exaggerated interventricular dependence, is characteristic of CP. Accordingly respiratory flow variations consisting in increasing >25% in mitral inflow during expiration and >40% in tricuspid inflow after inspiration are absent in RCM but frequently noted in CP ( 54 ). However, among all echocardiographic parameters, the most useful to distinguish RCM from CP are those of tissue Doppler imaging.…”

Section: Diagnosismentioning

confidence: 99%

Pediatric Restrictive Cardiomyopathies

et al. 2022

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Restrictive cardiomyopathy (RCM) is the least frequent phenotype among pediatric heart muscle diseases, representing only 2.5–3% of all cardiomyopathies diagnosed during childhood. Pediatric RCM has a poor prognosis, high incidence of pulmonary hypertension (PH), thromboembolic events, and sudden death, is less amenable to medical or surgical treatment with high mortality rates. In this scenario, heart transplantation remains the only successful therapeutic option. Despite a shared hemodynamic profile, characterized by severe diastolic dysfunction and restrictive ventricular filling, with normal ventricle ejection fraction and wall thickness, RCM recognizes a broad etiological spectrum, consisting of genetic/familial and acquired causes, each of which has a distinct pathophysiology and natural course. Hence, the aim of this review is to cover the causes, clinical presentation, diagnostic evaluation, treatment, and prognosis of pediatric RCM.

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Noninvasive Multimodality Imaging for the Diagnosis of Constrictive Pericarditis

Cited by 55 publications

References 62 publications

Constrictive Pericarditis: An Unusual Presentation of Rheumatoid Vasculitis

Constrictive Pericarditis: An Unusual Presentation of Rheumatoid Vasculitis

Discordant clinical presentation and pathophysiology: insights with cardiac magnetic resonance

Pediatric Restrictive Cardiomyopathies

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