Observations on a case of external pancreatic fistula in man

Sinclair, Ian

doi:10.1002/bjs.18004418506

Cited by 34 publications

(3 citation statements)

References 28 publications

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“…The observations of Kogen (1931) on 17 patients are open to criticism because gastric juice was not prevented from entering the duodenum: the increased pancreatic response after sham feeding may not be a true cephalic phase but secondary to the HCI produced during the cephalic phase of gastric secretion (Alphin and Lin, 1959). In a man with a pancreatic fistula neither Villaret and Justin-Besancon (1936) nor Sinclair (1956) were able to stimulate pancreatic secretion when appetizing food was shown to the patient.…”

mentioning

confidence: 99%

Cephalic phase of pancreatic secretion in man.

Sarles¹,

Dani²,

Prezelin³

et al. 1968

Gut

112

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mentioning

confidence: 99%

Cephalic phase of pancreatic secretion in man.

Sarles¹,

Dani²,

Prezelin³

et al. 1968

Gut

112

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“…The secretion of calcium into the duodenum in man has been studied by Hansky (1967) and from naturally occurring pancreatic fistulae by Kogut, Matzner, and Sobel (1936) and Sinclair (1956). Hansky (1967) reported an increased concentration of calcium in both the basal and the secretin-stimulated pancreatic juice in chronic pancreatitis although the total calcium output after stimulation was normal.…”

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confidence: 99%

The production of calcium and magnesium during pancreatic function tests in health and disease

Nimmo¹,

Finlayson²,

Smith³

et al. 1970

Gut

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SUMMARY Duodenal calcium and magnesium concentrations after pancreatic stimulation were measured in 25 patients with and without pancreatic disease. The concentration of both ions was higher in those with pancreatic disease, and furthermore these patients showed a different secretory pattern. These findings may be relevant to the aetiology of pancreatitis and to pancreatic calcification, and might be of diagnostic value.Pancreatic calcification is often seen in chronic pancreatitis and it is accepted that the pancreatic inflammatory disorders can be associated with abnormalities of calcium metabolism. For example between 7 and 19% of patients with hyperparathyroidism have had pancreatitis (Mixter,

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“…Miller and Ginsberg (1950) stated that ' trypsinogen' could not be obtained from the pancreatic juice. Garis and Merkel (1934) and Sinclair (1956) found that 'trypsin' was absent from the juice obtained from their patients with pancreatic 587 IN PANCREATICOBILIARY TREE fistulae. Lahey and Lium (1937) found active proteolytic enzymes, although the activity may have been due to contamination by duodenal contents.…”

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confidence: 96%