Obstruction of the Right Coronary Artery Ostium by an Aortic Valve Papillary Fibroelastoma

Granger, Emily; Rankin, Jamie; Larbalestier, Robert; Hockings, B.

doi:10.1016/j.hlc.2005.04.010

Cited by 13 publications

(15 citation statements)

References 9 publications

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“…The etiology of the ventricular arrhythmia was most likely to be scar‐related post myocardial infarction. However, a mechanism whereby ischemic VT due to intermittent obstruction of the right coronary artery ostium by the tumor is also possible 7,8 . Furthermore, it is possible that the inferolateral myocardial infarct was caused by embolization of material from the fibroelastoma rather than atherosclerotic plaque rupture, resulting in an occluded posterior left ventricular branch, since there were no angiographic appearances suggesting thrombus at the time of angiography.…”

Section: Discussionmentioning

confidence: 99%

“…However, a mechanism whereby ischemic VT due to intermittent obstruction of the right coronary artery ostium by the tumor is also possible. 7,8 Furthermore, it is possible that the inferolateral myocardial infarct was caused by embolization of material from the fibroelastoma rather than atherosclerotic plaque rupture, resulting in an occluded posterior left ventricular branch, since there were no angiographic appearances suggesting thrombus at the time of angiography. The history of recurrent neurological events over 6 years could represent multiple embolization events from a slow growing fibroelastoma.…”

Section: Figure 1 Transesophageal Echocardiogram (Short Axis View Atmentioning

confidence: 99%

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Cardiac Papillary Fibroelastoma Presenting with Recurrent Ventricular Tachycardia

Asrress

Mitchell²,

Evans

et al. 2007

Pacing Clinical Electrophis

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A 72-year-old man presented with multiple episodes of pulseless ventricular tachycardia, 10 days following an ST-segment elevation myocardial infarction. He had a 6-year cardiovascular history that included four neurological events and two myocardial infarctions. Transesophageal echocardiography revealed two mobile masses on the aortic valve. A transaortic surgical approach was successful in removing both masses. Histology confirmed papillary fibroelastoma. The patient subsequently received an implantable cardioverter defibrillator.

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Section: Discussionmentioning

confidence: 99%

Section: Figure 1 Transesophageal Echocardiogram (Short Axis View Atmentioning

confidence: 99%

Cardiac Papillary Fibroelastoma Presenting with Recurrent Ventricular Tachycardia

Asrress

Mitchell²,

Evans

et al. 2007

Pacing Clinical Electrophis

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“…Several potential etiologies or mechanisms underlying the occurrence of AMI in patients with near-normal coronary angiograms have been proposed, including concealed atherosclerosis, coronary spasm,8 thrombosis and hypercoagulability,5-7 embolisation,9,10 and inflammation,4 although the eventual etiology remains uncertain in the majority of patients. To determine whether the clinical characteristics and prognosis differed according to the underlying mechanisms, in the present study, we further classified the MINC cohort into 2 subgroups according to their causative pathology: Group II-a (cryptogenic) and Group II-b (with possible etiologies).…”

Section: Discussionmentioning

confidence: 99%

“…Myocardial infarction with angiographically near-normal coronary arteries (MINC) is a multifactorial syndrome for which the exact cause often remains undetermined in individual patients 2-4. Disparate etiologic factors have been incriminated in MINC, i.e., transient occlusion of the infarct-related artery owing to platelet hyperactivity and thrombosis (hypercoagulability),5-7 coronary artery spasm,8 coronary embolism,9,10 and coronary abnormalities such as myocardial bridges, coronary ectasia, or coronary dissection 11…”

Section: Introductionmentioning

confidence: 99%

Is Myocardial Infarction in Patients without Significant Stenosis on a Coronary Angiogram as Benign as Believed?

et al. 2012

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The present study aimed to investigate the clinical characteristics and 1-year outcomes of acute myocardial infarction (AMI) patients without significant stenosis on a coronary angiogram comparison with the clinical characteristics and outcomes of patients with significant coronary artery stenosis. A total of 1,220 patients with AMI were retrospectively classified into Group I (≥50% diameter stenosis, n=1,120) and Group II (<50%, n=100). Group II was further divided into two subgroups according to the underlying etiology: cryptogenic (Group II-a, n=54) and those with possible causative factors (Group II-b, n=46). Patients in Group II were younger, were more likely to be women, and were less likely to smoke and to have diabetes mellitus than were patients in Group I. The levels of cardiac enzymes, LDL-cholesterol levels, and the apo-B/A1 ratio were lower in Group II. However, 1-month and 12-month rates of major adverse cardiac events (MACE) were not significantly different between the two groups. The Group II-b subgroup comprised 29 patients with vasospasm, 11 with myocardial bridge, and 6 with spontaneous thrombolysis. Left ventricular ejection fraction and creatinine clearance were lower and levels of N-terminal pro-brain natriuretic peptide (NT-proBNP) and high-sensitivity C-reactive protein (hs-CRP) were higher in Group II-a than in Group II-b. However, outcomes including MACE and mortality at 12 months were not significantly different between the two subgroups. The 1-year outcomes of patients in Group II were similar to those of patients in Group I. The clinical outcomes in Group II-a were also similar to those of Group II-b, although the former group showed higher levels of NT-proBNP and hs-CRP.

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“…In previous studies, 1-12% of patients with AMI who underwent coronary angiography did not develop an irregular lumen, or coronary artery stenosis of <50% (3,4). In this type of AMI, causes of infarct-related artery occlusion vary from person to person, such as increased platelet or coagulation activity (hypercoagulability) (5-7), coronary artery spasm (8), coronary artery embolism (9,10), as well as coronary myocardial bridge, coronary artery ectasia and coronary artery dissection (11). AMI caused by coronary artery embolism is rare in clinical practice.…”

Section: Introductionmentioning

confidence: 98%

Clinical analysis of acute myocardial infarction caused by coronary embolism

et al. 2017

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IntroductionAcute myocardial infarction (AMI) is very common in clinic, and is often caused by incomplete coronary occlusion or complete occlusion induced by acute plaque rupture (1). Coronary angiography plays an important role in the etiology diagnosis and treatment of AMI (2). In previous studies, 1-12% of patients with AMI who underwent coronary angiography did not develop an irregular lumen, or coronary artery stenosis of <50% (3,4). In this type of AMI, causes of infarct-related artery occlusion vary from person to person, such as increased platelet or coagulation activity (hypercoagulability) (5-7), coronary artery spasm (8), coronary artery embolism (9,10), as well as coronary myocardial bridge, coronary artery ectasia and coronary artery dissection (11). AMI caused by coronary artery embolism is rare in clinical practice. This study conducts an analysis of the clinical characteristics of these patients. MethodsFrom 2005 to 2015, five patients with AMI caused by coronary artery embolism were admitted to our hospital, and underwent primary percutaneous coronary intervention Original Article

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Obstruction of the Right Coronary Artery Ostium by an Aortic Valve Papillary Fibroelastoma

Cited by 13 publications

References 9 publications

Cardiac Papillary Fibroelastoma Presenting with Recurrent Ventricular Tachycardia

Cardiac Papillary Fibroelastoma Presenting with Recurrent Ventricular Tachycardia

Is Myocardial Infarction in Patients without Significant Stenosis on a Coronary Angiogram as Benign as Believed?

Clinical analysis of acute myocardial infarction caused by coronary embolism

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