Ocular Bobbing: Abnormal Eye Movement or Eye Movement’s Abnormality?

Larmande, Pierre; Limodin, J; Hénin, Dominique; Lapierre, F

doi:10.1159/000309318

Cited by 7 publications

(8 citation statements)

References 8 publications

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“…The lesion was, indeed, a tuberous sclerosis-related hamartoma. The presence of an intrinsic pontine lesion and the preservation of horizontal movements is in contrast to the hypothesis of Larmande et al (1983).…”

Section: Figure 3: Magnetic Resonance Image (Mri) Showing Lesions Caumentioning

confidence: 61%

“…Vertical movements should be dependent on pontine lesions where the vestibular nucleus and vertical tracts are protected as these movements develop on the loss of horizontal movements. Larmande et al (1983) suggested that ocular bobbing should not be regarded as an abnormal eye movement, but as the residual movement of patients who are totally deprived of both horizontal and upward movements. Most types of bobbing movement generally develop as a result of pontine hemorrhage.…”

Section: Figure 3: Magnetic Resonance Image (Mri) Showing Lesions Caumentioning

confidence: 99%

“…It usually develops after intrinsic pontine lesions (Larmande et al 1983). However, the exact pathophysiological mechanism that causes bobbing movement is controversial (DaneshMeyer 2002).…”

mentioning

confidence: 99%

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Unilateral reverse ocular bobbing caused by tuberous sclerosis

Karakaş¹,

Yakıncı²,

Kemal³

et al. 2006

Develop Med Child Neuro

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A female aged 12 months with developmental delay and left‐sided reverse ocular bobbing (rapid deviation of the eye upward and a slow return to the horizontal position) was observed. At birth, an upper left gingival mass, pathologically diagnosed as a benign granular cell tumour, was removed. On computed tomography (CT), left middle cerebral pedincular and midpontine lesions were seen. Magnetic resonance imaging revealed additional white matter hamartomas, corticosubcortical tubers, and subependymal nodules. The patient was re‐examined at 36 months. Her general developmental quotient was equivalent to 23 months of age. Third cranial nerve functions and auditory brainstem response were normal. Her abnormal eye movements were still present. Reverse bobbing is usually observed in patients who are unconscious and who have significant pontine pathology and disruption of the reticular formation. This case is the first tuberous sclerosis‐related ocular bobbing case to our knowledge and is interesting as the causative lesion was relatively mild, and ocular horizontal movements were preserved.

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Section: Figure 3: Magnetic Resonance Image (Mri) Showing Lesions Caumentioning

confidence: 61%

Section: Figure 3: Magnetic Resonance Image (Mri) Showing Lesions Caumentioning

confidence: 99%

See 1 more Smart Citation

Unilateral reverse ocular bobbing caused by tuberous sclerosis

Karakaş¹,

Yakıncı²,

Kemal³

et al. 2006

Develop Med Child Neuro

View full text Add to dashboard Cite

show abstract

“…Vertical movements should be dependent on pontine lesions where the vestibular nucleus and vertical tracts are protected as these movements develop on the loss of horizontal movements. The presence of an intrinsic pontine lesion and the preservation of horizontal movements is in contrast to the hypothesis of Larmande et al (1983). Most types of bobbing movement generally develop as a result of pontine hemorrhage.…”

Section: Discussionmentioning

confidence: 86%

“…It usually develops after intrinsic pontine lesions (Larmande et al 1983). It usually develops after intrinsic pontine lesions (Larmande et al 1983).…”

mentioning

confidence: 99%

Unilateral reverse ocular bobbing caused by tuberous sclerosis

Karakaş¹,

Yakıncı²,

Fırat³

et al. 2006

Dev Med Child Neurol

View full text Add to dashboard Cite

A female aged 12 months with developmental delay and left-sided reverse ocular bobbing (rapid deviation of the eye upward and a slow return to the horizontal position) was observed. At birth, an upper left gingival mass, pathologically diagnosed as a benign granular cell tumour, was removed. On computed tomography (CT), left middle cerebral pedincular and midpontine lesions were seen. Magnetic resonance imaging revealed additional white matter hamartomas, corticosubcortical tubers, and subependymal nodules. The patient was re-examined at 36 months. Her general developmental quotient was equivalent to 23 months of age. Third cranial nerve functions and auditory brainstem response were normal. Her abnormal eye movements were still present. Reverse bobbing is usually observed in patients who are unconscious and who have significant pontine pathology and disruption of the reticular formation. This case is the first tuberous sclerosis-related ocular bobbing case to our knowledge and is interesting as the causative lesion was relatively mild, and ocular horizontal movements were preserved.

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Specificity of "Peering at the Tip of the Nose" for a Diagnosis of Thalamic Hemorrhage

Choi¹,

Jung²,

Kim³

2004

Arch Neurol

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Tonic inward and downward deviation of the eyes may develop in thalamic or brainstem lesions. Irritation or destruction of the neural structures involved in the vergence and vertical gaze may cause this ocular sign in mesodiencephalic lesions. Skew deviation and esotropia from abduction deficit may be involved in some patients. Ocular bobbing and tonic downward deviation may share a common pathogenesis.

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Ocular Bobbing: Abnormal Eye Movement or Eye Movement’s Abnormality?

Cited by 7 publications

References 8 publications

Unilateral reverse ocular bobbing caused by tuberous sclerosis

Unilateral reverse ocular bobbing caused by tuberous sclerosis

Unilateral reverse ocular bobbing caused by tuberous sclerosis

Specificity of "Peering at the Tip of the Nose" for a Diagnosis of Thalamic Hemorrhage

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