Oculosympathetic Paresis Associated by Cluster Headaches

Riley, Fenwick C.; Moyer, Norma J.

doi:10.1016/0002-9394(71)90015-8

Cited by 51 publications

(32 citation statements)

References 6 publications

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“…This is in agreement with findings reported independently by Nieman & Hurwitz (20) and Kunkle & Anderson (21) 20 years ago and later substantiated by others (22,25), that there is evidence of a sympathetic lesion in the iris of the symptomatic side. It is therefore not necessary to postulate a "parasympathetic overactivity" during attacks as far as the sweat glands are concerned.…”

Section: Values (G/m 2 /H) Cluster Headachesupporting

confidence: 93%

Cluster Headache: On the Mechanism Behind Attack-Related Sweating

Saunte¹,

Russell

Sjaastad³

1983

Cephalalgia

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Eighteen cluster headache patients were studied using body heating or exercise tests; all but two of them were also studied with a pilocarpine test (0.1 mg/kg body wt, s.c.). Evaporimeter measurements were made on both sides of the forehead under standard conditions in a thermo room. Heat- and exercise-induced sweating was clearly less pronounced on the symptomatic side than on the non-symptomatic side of the forehead, and was significantly different compared to controls. Pilocarpine on the other hand induced a clearly more pronounced response on the symptomatic side than on the non-symptomatic side, which was also statistically significantly higher than in the control group. These findings suggest a supersensitivity of the sweat glands to pilocarpine on the symptomatic side of the forehead in most cases of cluster headache.

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Section: Values (G/m 2 /H) Cluster Headachesupporting

confidence: 93%

Cluster Headache: On the Mechanism Behind Attack-Related Sweating

Saunte¹,

Russell

Sjaastad³

1983

Cephalalgia

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“…However, based on present knowledge regarding the neural control of cardiovascular function (5,6,7) and evidence of heart rate and ECG changes in other neurological conditions (29,30), the findings of this study suggest that there is a central disturbance in autonomic function in cluster headache (31). In addition, it is extremely difficult to propose a lesion in the peripheral autonomic nervous system which could explain not only these heart rate changes, but also the sweating pattern (8), and the ocular and nasal symptoms and signs (32)(33)(34)(35)(36)(37)(38)(39)(40)(41)(42) which are frequently present during attacks of this condition. Heart rate before 1 57 81 2 49 76 3 110 119, 125 4 65, 53 83 5 80, 78, 71 78 6 47, 52 62…”

Section: Resultsmentioning

confidence: 99%

Cluster Headache: A Computerized Analysis of 24 h Holter ECG Recordings and Description of ECG Rhythm Disturbances

Russell

Storstein

1983

Cephalalgia

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Continuous ECG recording has been carried out for at least 24 h in 27 patients suffering from cluster headache. During the study a total of 84 attacks occurred in 25 of the 27 patients who took part. A computerized analysis of the heart rate changes accompanying attacks showed the following: (i) an increase in heart rate at the onset of attacks; the degree of this increase being dependent on the heart rate before attacks, (ii) a relative decrease in heart rate and increased variations in heart rate during attacks, (iii) a relative increase in heart rate at the end of attacks, and (iv) a relative decrease in heart rate after attacks. Five patients (18.5%) showed ECG rhythm disturbances: two frequent premature ventricular beats, one transient attacks of atrial fibrillation, one first degree atrio-ventricular block, and one patient sinoatrial block.

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“…A transient postganglionic Horner syndrome can be seen in 5%–22% of these patients, and permanent oculosympathetic paresis has been reported in some patients who experience repeated attacks 66,67. Raeder paratrigeminal neuralgia, a painful postganglionic Horner syndrome accompanied by ipsilateral trigeminal neuralgia, is associated with lesions in the middle cranial fossa medial to the trigeminal ganglion 68–70…”

Section: Localizationmentioning

confidence: 99%

Horner syndrome: clinical perspectives

Kanagalingam¹,

Miller²

2015

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Horner syndrome consists of unilateral ptosis, an ipsilateral miotic but normally reactive pupil, and in some cases, ipsilateral facial anhidrosis, all resulting from damage to the ipsilateral oculosympathetic pathway. Herein, we review the clinical signs and symptoms that can aid in the diagnosis and localization of a Horner syndrome as well as the causes of the condition. We emphasize that pharmacologic testing can confirm its presence and direct further testing and management.

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Oculosympathetic Paresis Associated by Cluster Headaches

Cited by 51 publications

References 6 publications

Cluster Headache: On the Mechanism Behind Attack-Related Sweating

Cluster Headache: On the Mechanism Behind Attack-Related Sweating

Cluster Headache: A Computerized Analysis of 24 h Holter ECG Recordings and Description of ECG Rhythm Disturbances

Horner syndrome: clinical perspectives

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