Olecranon bursitis caused by group C streptococcus in a patient with tophaceous gout

Morsch, Claudine Predebon; Batistella, Fábio; Staub, Henrique Luiz

doi:10.1016/j.bjid.2012.08.024

Cited by 5 publications

(2 citation statements)

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“…They have shown a 100% increase of bacteremia between 2006 and 2012 and an increase of SDSE infections in Asia, Europe, and the USA [12, 14]. Of particular interest are the reports about the relations between GCS with the pathogenesis of rheumatic fever in Australia and India [5, 21, 23] and first documented zoonotic diseases [6, 8, 9, 22, 24]. …”

Section: Pathogenicitymentioning

confidence: 99%

“…The patient was a 24-year-old man with HLA-B27 positive genotype (this genotype predisposing an individual also to poststreptococcal reactive arthritis). Morsch, however, describes the case of a 72-year-old man, suffering from tophaceous gout, in whom olecranon bursitis developed as the result of a GCS infection [24]. The author notes that until now circa 15 cases of septic arthritis (GCS) have been documented, but none have been related to bursitis [24].…”

Section: Pathogenicitymentioning

confidence: 99%

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Pathogenicity of Virulent Species of Group C Streptococci in Human

Kłos

Wójkowska‐Mach

2017

Canadian Journal of Infectious Diseases and Medical Microbiolog

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Group C streptococci (GCS) are livestock pathogens and they often cause zoonotic diseases in humans. They are Gram-positive, in mostly β-hemolytic and facultative anaerobes. Because of their close evolutionary kinship with group A streptococci (GAS), GCS share many common virulence factors with GAS and cause a similar range of diseases. Due to the exchange of genetic material with GAS, GCS belong to bacteria that are difficult to be distinguished from group A streptococci; GCS are often treated in microbiological diagnostics as contamination of the culture. This report focuses mainly on the pathogenicity of virulent species of GCS and their association with human diseases. The condition that is most frequently quoted is pharyngitis. In this paper, the virulence factors have also been mentioned and an interesting link has been made between GCS and the pathogenesis of rheumatic diseases among the native people of India and Aboriginal populations.

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Section: Pathogenicitymentioning

confidence: 99%

Section: Pathogenicitymentioning

confidence: 99%

Pathogenicity of Virulent Species of Group C Streptococci in Human

Kłos

Wójkowska‐Mach

2017

Canadian Journal of Infectious Diseases and Medical Microbiolog

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Recent Developments in Septic Bursitis

Hanrahan

2013

Curr Infect Dis Rep

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Septic bursitis is a disease that can be difficult to distinguish from aseptic bursitis. There are no definitive treatment guidelines. The optimal duration of antibiotic therapy is not defined. Most of the cases are due to infection with Staphylococcus aureus, although many other pathogens can also cause this infection. The optimal management includes early recognition and prompt initiation of appropriate antibiotic therapy. Surgical management may be necessary, and complications can arise from open procedures. Endoscopic bursectomy may be considered for patients requiring surgical debridement. This approach may lead to decreased morbidity in those requiring surgical intervention.

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Bilateral olecranon bursitis – A rare clinical presentation of gout

Sargın

Gürer

2018

The Egyptian Rheumatologist

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Olecranon bursitis caused by group C streptococcus in a patient with tophaceous gout

Cited by 5 publications

References 3 publications

Pathogenicity of Virulent Species of Group C Streptococci in Human

Pathogenicity of Virulent Species of Group C Streptococci in Human

Recent Developments in Septic Bursitis

Bilateral olecranon bursitis – A rare clinical presentation of gout

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