Optic Disc Hemorrhages and Progression of Glaucoma

Siegner, Scott W.; Netland, Peter A.

doi:10.1016/s0161-6420(96)30572-1

Cited by 170 publications

(115 citation statements)

References 41 publications

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“…ONHH likely occurs due to the infarction of the blood supply to the ONH. [36][37][38][39][40] ONHH is observed rarely in normal eyes and appears more often in eyes with glaucoma. ONHH at the border of the optic disc is a highly pathognomonic finding of GON.…”

Section: -6122120-35mentioning

confidence: 99%

“…ONHH associated with early glaucoma is usually located on the supero-temporal and inferotemporal regions of the optic disc. [36][37][38][39][40] ONHH is also a strong indicator for the progression of glaucoma.…”

Section: -6122120-35mentioning

confidence: 99%

“…ONHH often recurs in the same area of the disc until a notch is formed, and then occurs at other areas of the disc where the rim is still normal. [36][37][38][39][40] ONHH is more common in patients with large IOP variations. ONHH typically resolves within 6-10 weeks of onset and is associated with localized RNFL defects and NRR notching.…”

Section: -6122120-35mentioning

confidence: 99%

“…ONHH is usually missed in clinical observation because of its resolving time. [36][37][38][39][40] Non-glaucomatous ONHH can occur in patients with posterior vitreous detachment, branch retinal vein occlusion, diabetic retinopathy, hypertensive retinopathy, AION, and in patients with anaemia.…”

Section: -6122120-35mentioning

confidence: 99%

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Pearls for Correct Assessment of Optic Disc at Glaucoma Diagnosis

Turgut¹

2017

US Ophthalmic Review

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The correct evaluation of the optic disc, and related structures in ophthalmoscopy, is critical for the diagnosis of open angle glaucoma because usually glaucomatous optic nerve damage firstly occurs in the optic disc before detectable visual field defects become apparent; however, some studies have demonstrated that visual field defects can also be found without any glaucomatous changes in the optic disc. The purpose of this review is to provide a guide to clinicians for the evaluation of the optic disc in glaucoma suspects and patients. This guide can facilitate the diagnosis and exclusion of glaucoma in these subjects.

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Section: -6122120-35mentioning

confidence: 99%

Section: -6122120-35mentioning

confidence: 99%

Section: -6122120-35mentioning

confidence: 99%

Section: -6122120-35mentioning

confidence: 99%

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Pearls for Correct Assessment of Optic Disc at Glaucoma Diagnosis

Turgut¹

2017

US Ophthalmic Review

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“…In most materials hh are seldom seen. As few as one per cent among glaucoma patients are given in a recent report by Siegner & Netland (1996). On the other hand, there are reports where the probability of finding a haemorrhage (h) in a newly detected manifest glaucoma case is 25-35 per cent (Bengtsson, personal communication 1998).…”

Section: Introductionmentioning

confidence: 99%

Spotting of disc haemorrhages on fundus pictures; on Kodachrome slides and on digitally enhanced images

Holm

Krakau

2000

Acta Ophthalmologica Scandinavica

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ABSTRACT.Purpose: To find out whether digital processing can facilitate interpretation of fundus photographs. Methods: Digitised fundus pictures were manipulated and enhanced by a process called histogram equalisation. Essentially, the gamma values for red (R), green (G), and blue (B) were separately modified. The principle is explained and exemplified. Material and Results: Small disc haemorrhages, which may normally be hard to detect, became more visible after histogram equalisation, which is demonstrated in test series. Conclusion: Digital histogram equalisation offers a new way of looking at fundus pictures, which, alone or combined with other capabilities of digital processing, has great potential for diagnosis, as well as for follow up studies. The possibility to decide the true nature of suspect spots after applying the enhancement procedure is likewise of great value.

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Photodynamic Therapy With Verteporfin for Choroidal Neovascularization Caused by Age-related Macular Degeneration

Schmidt‐Erfurth¹

1999

Arch Ophthalmol

305

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To evaluate safety and short-term visual acuity and fluorescein angiographic effects of photodynamic therapy (PDT) after retreatments with verteporfin for choroidal neovascularization (CNV) in agerelated macular degeneration (AMD) that demonstrated fluorescein leakage after at least 1 course of PDT. Design: Nonrandomized, multicenter, open-label phase 1 and 2 clinical trial using 2 different retreatment dosage regimens. Setting: Four ophthalmic centers in Europe and North America providing retinal care. Methods: Standardized protocol refraction, visual acuity testing, ophthalmic examinations, color photographs, and fluorescein angiograms were used to evaluate the results of multiple PDT treatments. Two regimens (regimens 2 and 4) for treatment and retreatment were chosen from 5 used in a single-treatment study. Both regimens used a verteporfin dose of 6 mg/m 2 infused for 10 minutes. However, regimen 2 used a light dose of 100 J/cm 2 applied 20 minutes after the start of the verteporfin infusion, whereas regimen 4 used a light dose of 50, 75, or 100 J/cm 2 applied 15 minutes after infusion commenced. Posttreatment evaluations were planned in 31 participants up to 3 months after up to 2 retreatments given at 2-or 4-week intervals after initial PDT treatment. Similar posttreatment evaluations were planned after retreatments in 5 additional participants who were reenrolled some time more than 12 weeks after an initial PDT treatment. Results: The average visual acuity change for the 31 participants who had retreatment within 2 to 4 weeks after

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Optic Disc Hemorrhages and Progression of Glaucoma

Cited by 170 publications

References 41 publications

Pearls for Correct Assessment of Optic Disc at Glaucoma Diagnosis

Pearls for Correct Assessment of Optic Disc at Glaucoma Diagnosis

Spotting of disc haemorrhages on fundus pictures; on Kodachrome slides and on digitally enhanced images

Photodynamic Therapy With Verteporfin for Choroidal Neovascularization Caused by Age-related Macular Degeneration

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