Orbital haemorrhage with loss of vision in a patient with disseminated intravascular coagulation and prostatic carcinoma

Goyal, Saurabh; Goel, Ravi

doi:10.1080/01676830490504197

Cited by 8 publications

(2 citation statements)

References 6 publications

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“…22,23 However, drug therapy or hemorrhage due to congenital or systemic conditions may lead to nontraumatic RBH. [24][25][26][27] Drug-related nontraumatic RBH occurs in hemorrhagic diatheses due to circulating anticoagulation medication, whereas vessel malformations are congenital causes of RBH. [28][29][30] RBH can occur spontaneously in drug-related pharmacological or congenital conditions.…”

Section: Retrobulbar Hemorrhagementioning

confidence: 99%

Diagnosis and Treatment of Optic Nerve Trauma

et al. 2014

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Decreasing visual acuity secondary to orbital trauma or orbital and anterior skull base surgery may be caused by either sudden space-occupying intraorbital lesions, including retrobulbar hemorrhage (RBH), or direct damage to the prechiasmatic pathway. Contrary to traumatic optic neuropathy, RBH must be diagnosed and treated immediately to prevent permanent damage to the visual system. Therefore, monitoring and handling of visual pathway damage are mandatory. Flash visual evoked potentials and electroretinograms can provide evidence of the status of conductivity of the visual pathway when clinical assessment is not feasible. Both are thus essential diagnostic procedures not only for primary diagnosis but also for intraoperative evaluation. In case of RBH surgical decompression is compulsory. However, traumatic optic neuropathy does not respond to either corticosteroids or optic canal surgery. Modern craniomaxillofacial surgery requires detailed consideration of the diagnosis and treatment of traumatic visual pathway damage with the ultimate goal of preserving visual acuity.

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Section: Retrobulbar Hemorrhagementioning

confidence: 99%

Diagnosis and Treatment of Optic Nerve Trauma

et al. 2014

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“…13,14 In nonpenetrating orbital trauma, fractures of the orbit following blunt facial trauma may be associated with RBH, and loss of vision occurs in 0.3% of these patients. 15,16 However, drug therapy 17 or hemorrhage due to congenital or systemic conditions [18][19][20] may lead to nontraumatic RBH. Drug-related nontraumatic RBH occurs in hemorrhagic diatheses due to circulating anticoagulation medication, 21,22 whereas vessel malformations are congenital causes of RBH.…”

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confidence: 99%

Efficacy of Transcutaneous Transseptal Orbital Decompression in Treating Acute Retrobulbar Hemorrhage and a Literature Review

Zimmerer

Schattmann

Essig

et al. 2014

Craniomaxillofacial Trauma & Reconstruction

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EtiologyRetrobulbar hemorrhage (RBH) is an uncommon but potentially blinding orbital emergency in which immediate diagnosis and treatment are necessary to maintain or reconstitute visual acuity. The literature indicates that two main causes of RBH can be distinguished as: traumatic and nontraumatic. Traumatic causes of RBH can be further classified into nonpenetrating and penetrating trauma. Penetrating orbital trauma is essentially iatrogenic. The most common causes AbstractDecreasing visual acuity secondary to orbital trauma may be caused by sudden spaceoccupying or expanding intraorbital lesions, including retrobulbar hemorrhage (RBH), herniation, or swelling. RBH must be diagnosed and treated immediately. This article addresses the efficacy of transcutaneous transseptal orbital decompression in a combination with a systematic review of the literature for a comparison of this method with existing treatment options. For this study the department's database was retrospectively screened for patients with acute RBH who were treated between 2009 and 2011 using the authors' approach. Patients presenting with RBH were classified into RBH classes I to III according to three different clinical and radiological manifestations of acute RBH. The efficacy of transcutaneous transseptal orbital decompression was assessed by postoperative visual acuities. The literature review was performed by using the MEDLINE database. The time period for the study was between 2009 and 2011 during which 10 patients were diagnosed with suspected RBH and 9 were treated with the authors' technique. Visual acuities were reconstituted or maintained in almost 86% of patients who were diagnosed and treated according to the authors approach and who survived initial trauma. It was concluded that transcutaneous transseptal orbital decompression provides an efficient and rapid approach for treating patients with acute RBH. By distinguishing three different manifestations of acute RBH, the authors present a diagnostic tool that may facilitate classification of RBH and determination of treatment options.

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