“…3 Histology can confirm the diagnosis by revealing irregular dysplastic endothelial channels, with a dense stroma containing smooth muscle, haemosiderin-laden macrophages, and lymphocytes. 3,4 In this case, it is difficult to elucidate whether ocular involvement is due to a primary OVLM or secondary to extension. If it is due to primary involvement, an earlier ocular presentation might be expected, although it is possible that the decrease in left visual acuity is related to a long-standing OVLM, rather than 'true amblyopia'.…”