There has been sustained interest in the importance of the P-wave duration in predicting a predisposition to atrial arrhythmias. 1-7 Many factors such as left atrial enlargement, left atrial hypertension, and altered conduction may affect P-wave duration. 11 P-wave duration has been shown to be highly correlated with intratrial conduction times. 12 Prolongation of intratrial and interatrial conduction times and the inhomogeneous propagation of sinus impulses are well-known electrophysiologic characteristics of the atrium prone to fibrillation. 13 Many studies have suggested that atrial fibrillation results from focal activation or multiple reentrant circuits, which necessitate areas of delayed conduction to initiate and maintain the rhythm. 14,15 The current feeling is that atrial fibrillation is usually imitated in the pulmonary veins and conduction delays would help maintain the arrhythmia. It is noteworthy that increased P-wave duration has been associated with many different clinical conditions. [16][17][18][19][20][21][22][23][24][25][26][27][28][29][30][31] In addition to direct measurement of P-wave duration on the standard electrocardiogram, several different methods of determining P-wave duration have been suggested. P-wave signal averaging, which requires special apparatus, has been used prospectively to identify patients at risk for developing atrial fibrillation. 8 With 12-lead surface electrocardiograms adjusted to 50 mm/second, the shortest and largest P-wave durations can be measured and the difference (P-wave dispersion) calculated. It has been shown that maximum P-wave duration and P-wave dispersion are significantly increased in individuals with a history of paroxysmal AF. 9 Another precise method of measuring P-wave duration uses six channel ECG recordings and a Address for reprints