P128 Gene expression profile in laryngeal cancer by oligonucleotide microarrays analysis

Markowski, Julia; Gierek, T; Oczko-Wojciechowska, Małgorzata; Kowalska, M.; Paluch, Jarosław; Jarząb, Michał; Wygoda, Zbigniew; Jarząb, Barbara; Wardas, Piotr; Lange, D

doi:10.1016/s1744-7895(07)70407-8

Cited by 17 publications

(18 citation statements)

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“…A high-density oligonucleotide microarray analysis of 14 squamous cell laryngeal carcinomas and matched adjacent normal mucosal controls revealed KIF14 overexpression as a molecular marker for laryngeal carcinomas [ 49 ]. A follow-up study of 24 samples confi rmed this fi nding [ 50 ].…”

Section: Kif14 Expression As a Tumor Marker And Prognostic Indicator mentioning

confidence: 84%

Kif14: A Clinically Relevant Kinesin and Potential Target for Cancer Therapy

Thériault

Corson

2015

Kinesins and Cancer

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Section: Kif14 Expression As a Tumor Marker And Prognostic Indicator mentioning

confidence: 84%

Kif14: A Clinically Relevant Kinesin and Potential Target for Cancer Therapy

Thériault

Corson

2015

Kinesins and Cancer

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“…KIF14 mRNA is overexpressed in retinoblastomas, in TAg‐RB and in many other cancers . Expression correlates with poor prognosis in breast, lung and ovarian cancers .…”

Section: Recurrent Gains and Losses Reveal Candidate Oncogenes And Tumentioning

confidence: 99%

The genomic landscape of retinoblastoma: a review

Thériault

Dimaras

Gallie

et al. 2013

Clinical Exper Ophthalmology

166

131

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Retinoblastoma is a paediatric ocular tumour that continues to reveal much about the genetic basis of cancer development. Study of genomic aberrations in retinoblastoma tumours has exposed important mechanisms of cancer development, and identified oncogenes and tumour suppressors that offer potential points of therapeutic intervention. The recent development of next-generation genomic technologies has allowed further refinement of the genomic landscape of retinoblastoma at high resolution. In a relatively short period of time, a wealth of genetic and epigenetic data has emerged on a small number of tumour samples. These data highlight the inherent molecular complexity of this cancer, despite the fact that most retinoblastomas are initiated by the inactivation of a single tumour suppressor gene. Here, we review the current understanding of the genomic, genetic and epigenetic changes in retinoblastoma, highlighting recent genome-wide analyses that have identified exciting candidate genes worthy of further validation as potential prognostic and therapeutic targets.

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“…Clearly, the mechanistic action of FOXN3 in mammalian cells needs further elucidation. In addition, despite the observations that the expression of FOXN3 is dysregulated in tumors from a variety of tissue origins (7,(12)(13)(14)(15)(16)(17)(18), the mechanistic involvement of FOXN3 in tumorigenesis remains to be investigated, and whether and how FOXN3 plays a role in the development and progression of breast cancer are currently unknown.…”

Section: Introductionmentioning

confidence: 99%