P2 Receptor-mediated Inhibition of Vasopressin-stimulated Fluid Transport and cAMP Responses in AQP2-transfected MDCK Cells

Kim, Yang Hoo; Choi, Young Jin; Bae, Hae Rahn; Woo, Jae Suk

doi:10.4196/kjpp.2009.13.1.9

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Distal Tubules1

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2013

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“…The proto-oncoprotein SYT(SS18) controls ATP release and regulates cyst formation by polarised MDCK cells, suggesting that SYT plays a vital role in controlling epithelial morphogenesis and might explain the lethality of its loss in the developing embryo [56]. P2 receptormediated inhibition of vasopressin (AVP)-stimulated fluid transport and cAMP response in AQP2-transfected MDCK cell has been reported [186].…”

Section: Distal Tubulesmentioning

confidence: 99%

Purinergic signalling in the kidney in health and disease

Burnstock

Evans

Bailey

2013

Purinergic Signalling

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The involvement of purinergic signalling in kidney physiology and pathophysiology is rapidly gaining recognition and this is a comprehensive review of early and recent publications in the field. Purinergic signalling involvement is described in several important intrarenal regulatory mechanisms, including tuboglomerular feedback, the autoregulatory response of the glomerular and extraglomerular microcirculation and the control of renin release. Furthermore, purinergic signalling influences water and electrolyte transport in all segments of the renal tubule. Reports about purine-and pyrimidine-mediated actions in diseases of the kidney, including polycystic kidney disease, nephritis, diabetes, hypertension and nephrotoxicant injury are covered and possible purinergic therapeutic strategies discussed.

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Section: Distal Tubulesmentioning

confidence: 99%