Pancreatic morphology a combined radiological and pathological study

Kreel, L.; Sandin, Brenda; Slavin, G

doi:10.1016/s0009-9260(73)80074-1

Cited by 51 publications

(15 citation statements)

References 4 publications

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“…The significance of the minimal-change pancreatitis noticed in the two noninsulin-dependent diabetics is dubious, as similar changes have been dem onstrated in normal people over the age of 50 years [10].…”

Section: Discussionsupporting

confidence: 59%

Endoscopic Retrograde Cholangiopancreatography in Sudanese Diabetics

Fedail¹,

Mahadi²,

Lidir³

et al. 1986

Digestion

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Endoscopic retrograde cholangiopancreatography (ERCP) was performed in 40 Sudanese patients with diabetes mellitus. 25 were insulin-dependent and 15 were noninsulin-dependent. ERCP was normal in all the patients with insulin-dependent diabetes. Two patients with noninsulin-dependent diabetes had pancreatographic changes compatible with minimal-change pancreatitis. Diabetes mellitus in Sudan unlike in other tropical countries is not associated with structural changes in the exocrine pancreas.

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Section: Discussionsupporting

confidence: 59%

Endoscopic Retrograde Cholangiopancreatography in Sudanese Diabetics

Fedail¹,

Mahadi²,

Lidir³

et al. 1986

Digestion

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“…For example, stenosis of the main duct is seen in carcinoma, chronic pancreatitis, or hyperplasia of the duct epithelium. These are associated with abnormalities in the parenchyma, but if this is normal then stenosis of the main duct may be due to compression by the superior mesenteric artery, aortic aneurysm, or large anterior osteophytes from the supine [2][3][4]. Obstruction of the main duct is found in carcinoma, chronic pancreatitis, or cyst [5].…”

Section: Discussionmentioning

confidence: 99%

Angiographic evaluation of the abnormal endoscopic pancreatogram

Ariyama

Shirakabe

et al. 1979

Gastrointest Radiol

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Out of 1,269 pancreatograms, 122 were abnormal. Angiography was performed in these patients. Fifty-five were found to have pancreatic carcinoma. In the remaining 67 patients a false positive angiographic diagnosis of either chronic pancreatitis or pancreatic cancer was made in 11%. In one patient a hemangioma was diagnosed as a pancreatic cyst. The remaining 58 patients all had normal pancreatic angiograms in spite of gross ductal abnormality on endoscopic retrograde cholangiopancreatography (ERCP). All these patients were followed for an average of 19 months and showed no clinical evidence of pancreatic disease. It is suggested that angiography should be considered a complementary examination to ERCP and is particularly useful to exclude carcinoma when the pancreatogram is abnormal.

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“…Accordingly, the reported patency of the APD of the typical pancreatic duct system (excluding cases of unfused pancreatic duct systems) has ranged widely, from 12% to 82% (Table 1). [2][3][4][5][6][7][8][9][10][11][12][13][14]37 Recently, some endoscopic retrograde pancreatography (ERP) studies have evaluated patency of the APD by observing the leakage of contrast medium from the MIP on fluoroscopy or radiography. 11,12,14 However, in many cases, the orifice of the MIP is very small and precise evaluation of patency of the APD is practically impossible using these methods.…”

Section: Patency Of the Apdmentioning

confidence: 99%

“…Patency of the APD has been assessed by direct injection of material into the pancreatic duct in resected or autopsy speciments. [2][3][4][5][6][7][8][9][10] Recently, endoscopic studies on patency of the APD have been performed, [11][12][13][14] and the clinical significance of the APD has been elucidated.…”

Section: Introductionmentioning

confidence: 99%

Clinical significance of the minor duodenal papilla and accessory pancreatic duct

Kamisawa

2004

J Gastroenterol

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The accessory pancreatic duct (APD) is the main drainage duct of the dorsal pancreatic bud in the embryo, entering the duodenum at the minor duodenal papilla (MIP). As development progresses, the duct of the dorsal bud undergoes varying degrees of atrophy at the duodenal end. In cases of patent APD, smooth-muscle fiber bundles derived from the duodenal proper muscular tunics surround the APD. The APD shows long and short patterns on pancreatography, and ductal fusion in the two types appears to differ embryologically. Patency of the APD in control cases, as determined by dye-injection endoscopic retrograde pancreatography, was 43%. Patency of the APD may depend on duct caliber, course, and terminal shape of the APD. A patent APD may prevent acute pancreatitis by reducing the pressure in the main pancreatic duct. Pancreas divisum is a common anatomical anomaly in which the ventral and dorsal pancreatic ducts do not unite embryologically. As the majority of exocrine flow is routed through the MIP in individuals with pancreas divisum, interrelationships between poor function of the MIP and increased flow of pancreatic juice caused by alcohol or diet may increase dorsal pancreatic duct pressure and lead to the development of pancreatitis. Wire-guided minor sphincterotomy, followed by dorsal duct stenting, is recommended for acute recurrent pancreatitis associated with pancreas divisum.

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Pancreatic morphology a combined radiological and pathological study

Cited by 51 publications

References 4 publications

Endoscopic Retrograde Cholangiopancreatography in Sudanese Diabetics

Endoscopic Retrograde Cholangiopancreatography in Sudanese Diabetics

Angiographic evaluation of the abnormal endoscopic pancreatogram

Clinical significance of the minor duodenal papilla and accessory pancreatic duct

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