2017
DOI: 10.1007/s11739-017-1634-8
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Parapharyngeal abscess with tracheal deviation

Abstract: A 41-year-old man presented to the Emergency Department (ED) with right-sided neck pain and swelling over the prior 48 h. He had a history of poor dentition and recent dental extractions. He was taking clindamycin for the infection and hydrocodone for pain. On examination, the patient was febrile and slightly confused. He had a hoarse voice, difficulty handling secretions, and significant induration and erythema of the right side of the face which extended down into the neck.

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Cited by 2 publications
(2 citation statements)
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“…Treatment of pharyngeal abscesses includes high-dose, IV antibiotic therapy covering both gram-positive aerobes and anaerobes [19] . Appropriate antibiotic choices are penicillins with beta-lactamase inhibitors (eg piperacillin-tazobactam, ampicillin-sulbactam), clindamycin, or high dose penicillin with metronidazole (as penicillin monotherapy would be insufficient coverage) [20] . Incision and drainage by an otolaryngologist is often required for complete resolution of these infections based on the extent of the abscess and degree of airway compromise [ 19 , 20 ].…”
Section: Discussionmentioning
confidence: 99%
“…Treatment of pharyngeal abscesses includes high-dose, IV antibiotic therapy covering both gram-positive aerobes and anaerobes [19] . Appropriate antibiotic choices are penicillins with beta-lactamase inhibitors (eg piperacillin-tazobactam, ampicillin-sulbactam), clindamycin, or high dose penicillin with metronidazole (as penicillin monotherapy would be insufficient coverage) [20] . Incision and drainage by an otolaryngologist is often required for complete resolution of these infections based on the extent of the abscess and degree of airway compromise [ 19 , 20 ].…”
Section: Discussionmentioning
confidence: 99%
“…Airway management is challenging in patients presenting with parapharyngeal abscess. First, medial bulging of the pharyngeal wall due to deep space pathology and edema from soft tissue inflammation distorts airway anatomy [9], modifies tissue planes [4], decreases tissue mobility [10], and impairs visualization and localization of the glottis [6]. Second, trismus, decreased interdental gap, and narrowing of the oropharyngeal isthmus [4] limit accessibility for intubation and subsequent administration of general anesthesia might conversely trigger airway closure and prevent intubation [11,12].…”
Section: Introductionmentioning
confidence: 99%