“…A variety of conditions can cause acquired hemichorea-hemiballismus syndrome. [ 9 – 11 ] These include structural damage to deep brain structures (cerebrovascular disease, [ 12 ] infection, trauma, neoplasia), or be associated with autoimmune/inflammatory disorders (antiphospholipid antibody syndrome, [ 13 ] paraneoplastic chorea), [ 14 ] metabolic derangement (nonketotic hyperglycemia), [ 15 , 16 ] or exposure to certain drugs (dopamine agonist or phenytoin). As shared with the same pattern of movements regardless of its underlying etiology, the differential diagnosis of hemichorea-hemiballismus syndrome relies on accompanying features, age, mode of onset and laboratory or imaging examinations.…”