Air Pollution - A Comprehensive Perspective 2012
DOI: 10.5772/51363
|View full text |Cite
|
Sign up to set email alerts
|

Particulate Air Pollutants and Respiratory Diseases

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1
1
1
1

Citation Types

0
16
0

Year Published

2015
2015
2024
2024

Publication Types

Select...
7
3

Relationship

0
10

Authors

Journals

citations
Cited by 19 publications
(16 citation statements)
references
References 102 publications
0
16
0
Order By: Relevance
“…This was contrary to the expected trend since respiratory disorders are usually more prevalent in urban communities due to increased exposure to transport and industrial atmospheric pollution emissions compared to rural areas (Jang, 2012;Gartner et al, 2008;O'Reilly et al, 2007;Iversen et al, 2005). Smoking is also a main cause of respiratory disease; however, in Mexico, the frequency of smoking is greater in urban (38.8%) compared to rural (23.7%) populations (CONADIC, 2008) (Hodzic et al, 2012;Li et al, 2012;Kodros et al, 2016).…”
Section: Respiratory Disease In Huejutla and Surrounding Areasmentioning
confidence: 93%
“…This was contrary to the expected trend since respiratory disorders are usually more prevalent in urban communities due to increased exposure to transport and industrial atmospheric pollution emissions compared to rural areas (Jang, 2012;Gartner et al, 2008;O'Reilly et al, 2007;Iversen et al, 2005). Smoking is also a main cause of respiratory disease; however, in Mexico, the frequency of smoking is greater in urban (38.8%) compared to rural (23.7%) populations (CONADIC, 2008) (Hodzic et al, 2012;Li et al, 2012;Kodros et al, 2016).…”
Section: Respiratory Disease In Huejutla and Surrounding Areasmentioning
confidence: 93%
“…In terms of mechanistic associations, PM 2.5 induces the release of inflammatory cytokines and the generation of free radicals, which may lead to oxidative stress, exacerbating some respiratory symptoms. 32 NO 2 -induced effects in the lung involve direct as well as free-radical-mediated oxidation of biomolecules, resulting in damage to the membrane structures, disruption of cellular metabolism and cell damage or death 33 . Potential mechanisms by which PM 2.5 is associated with cardiovascular events include the activation by particulate matter of inflammatory pathways, production of reactive oxygen species, alterations in vascular tone, and decreased heart rate variability (a marker of cardiac autonomic dysfunction) 34 .…”
Section: Methodsmentioning
confidence: 99%
“…There is now a wealth of evidence linking exposure to outdoor and indoor air pollution, especially airborne PMs, to adverse effects on allergic diseases, where PM's surface materials may target immune regulatory and pulmonary resident cells. 22,23 Several reviews have discussed the potential mechanisms of each individual component associated with PMs; for example, the functional impact of air pollutant gases such as O 3 , NO2, and CO has been extensively studied and several recent reviews 24,25 have proposed a series of mechanistic events resulting from PM exposure; their surface chemical and metal components have been used to establish the exposure-disease mechanistic relationship and explain the relationship between particulate air pollutants and allergic diseases. This study showed that levels of atmospheric pollutants O 3 , SO 2 , and PM10 increased by 10 μg/m 3 , the number of patient visits would increased by 1.95%, 1.19% and 0.33%, respectively ( Table 3, Figure 3), suggesting that air pollution might increase risk of AR onset.…”
Section: Discussionmentioning
confidence: 99%