Passive smoking and respiratory symptoms in the FinEsS Study

Larsson, Matz; Loit, Helle‐Mai; Meren, Mari; Pölluste, Jaak; Magnússon, Andrés; Larsson, K; Lundbäck, Bo

doi:10.1183/09031936.03.00033702

Cited by 90 publications

(91 citation statements)

References 28 publications

(31 reference statements)

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“…This found that the prevalence of respiratory symptoms remained higher in ex-smokers for many years compared with nonsmokers [29]. Passive smoking is associated with asthma and respiratory symptoms [17,30], and it is disappointing that data about passive smoking were not available. However, smoking was more common among manual workers, which is why passive smoking was probably also more common in this group and could contribute to the increased risk of asthma and respiratory symptoms.…”

Section: Socio-economic Status and Asthmamentioning

confidence: 95%

“…The questionnaire was developed from a revised version [15] of the UK Medical Research Council questionnaire [16], and has been used in other Swedish and international studies [17]. The questions about respiratory symptoms and diseases included the following: recurrent wheeze, attacks of shortness of breath, long-standing cough, sputum production, chronic productive cough, asthma, allergic rhinitis, chronic bronchitis or emphysema, and use of asthma medicines.…”

Section: Methodsmentioning

confidence: 99%

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Socio-economic status is related to incidence of asthma and respiratory symptoms in adults

Hedlund

Eriksson²,

Rönmark³

2006

Eur Respir J

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The aim of the present study was to assess the association between socio-economic status and impaired respiratory health in a 10-yr follow-up of a population-based postal survey in Northern Sweden.Multiple logistic regression was used to estimate odds ratios in relation to socio-economic class, using age, sex, a family history of asthma, smoking habits, and occupational exposures to dust, gases and fumes as possible confounders.The study comprised 2,341 males and 2,413 females. Cumulative incidences were generally lowest in professionals, including executives and civil servants at intermediate and higher levels, who were chosen as reference group. Manual workers in industry showed a significantly increased risk of developing asthma, recurrent wheeze, attacks of shortness of breath or a combination of the two, and chronic productive cough. Manual workers in service showed a similar pattern for attacks of shortness of breath, recurrent wheeze, or a combination of the two, and chronic productive cough. The corresponding population attributable risks were ,10%.Low socio-economic status was a risk factor for the development of asthma, symptoms common in asthma and chronic productive cough.

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Section: Socio-economic Status and Asthmamentioning

confidence: 95%

Section: Methodsmentioning

confidence: 99%

Socio-economic status is related to incidence of asthma and respiratory symptoms in adults

Hedlund

Eriksson²,

Rönmark³

2006

Eur Respir J

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“…

We read with interest the paper by LARSSON et al [1] in which they reported in a large population-based sample of nonsmokers that respiratory symptoms were related to environmental tobacco smoke (ETS). They gave special consideration to respiratory symptoms triggered by a variety of lower airway irritants and they showed for these symptoms that the relation was stronger in females than in males.

…”

mentioning

confidence: 72%

Sex differences in respiratory symptoms: Fig. 1.—

Varraso¹,

Oryszczyn²,

Kauffmann³

2003

Eur Respir J

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We read with interest the paper by LARSSON et al. [1] in which they reported in a large population-based sample of nonsmokers that respiratory symptoms were related to environmental tobacco smoke (ETS). They gave special consideration to respiratory symptoms triggered by a variety of lower airway irritants and they showed for these symptoms that the relation was stronger in females than in males. Although, such symptoms are particularly frequent among asthmatics [2], they did not perform an analysis according to asthma.As the epidemiological study on the genetics and environment of asthma (EGEA) includes a large number of adult asthmatics, as well as controls [3], we analysed whether sex differences were observed for symptoms triggered by various stimuli in 366 population-based (mainly electoral roll) controls and in 213 asthmatics cases. Triggers were exposure to hay/flowers, pets, dust, cold air, exercise and ETS. Symptoms were as in the study by LARSON et al.[1] respiratory symptoms (fits of coughing, shortness of breath and/or wheezing), but also nasal symptoms (runny nose and/or sneezes) and ocular symptoms (runny/itchy eyes) [4,5]. As expected, reports of symptoms triggered by various stimuli were markedly higher in asthmatics than in controls (for example respiratory symptoms triggered by ETS and pets were reported by 57% and 26% of the asthmatics and by 19% and 2% of the controls respectively). As in the LARSON et al.[1] study, females reported more symptoms than males in population-based controls (even when excluding asthmatics). Furthermore, considering asthmatic cases, a similar pattern was observed with females reporting in general more symptoms to any trigger than males. For respiratory symptoms, prevalences were generally higher in females than in males ( fig. 1). Age-adjusted odds ratios (ORs) were statistically significant for symptoms triggered by ETS and by exercise (ageadjusted OR (95% confidence interval (CI)) 2.4 (1.3-4.4) and 2.7 (1.3-5.6) respectively). The prevalence of nasal symptoms triggered by stimuli varied between 7% for exercise to 54% for dust in asthmatics. Females also reported more nasal symptoms than males for all triggers except hay/flowers, with age-adjusted ORs for ETS, exercise, pets and hay/flowers of 1.9 (0.9-4.0), 1.9 (0.6-5.8), 1.7 (0.9-3.4) and 1.0 (0.5-1.7) respectively.The analysis by LARSON et al.[1] was restricted to nonsmokers due to the focus on ETS. Considering sex differences in ETS-triggered respiratory symptoms according to active smoking is also of interest, as active smoking is related to the number of asthma attacks, an aspect of the severity of asthma [3]. Indeed, active smoking modified the association of sex with ETS-triggered symptoms. In nonsmokers and exsmokers, females reported more symptoms (age adjusted OR (95% CI) 2.5 (1.0-5.8) and 2.6 (0.8-8.1)), whereas no sex difference was observed in smokers (0.9 (0.2-5.4)).Our results confirm and extend the results of LARSON et al. [1]. The perception of symptoms triggered by various stimuli is recognised ...

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“…Una proporción de casos de EPOC ocurre en personas que no han fumado nunca. Entre personas no fumadoras (54) el tabaquismo pasivo es un factor de riesgo para el desarrollo de la EPOC (55)(56)(57)(58). Un estudio elaborado en niños de entre 6 y 18 años gallegos, donde el 56% de ellos estaban expuestos al humo del tabaco de alguno de sus progenitores, mostro un 75 % tenían reducida la capacidad espiratoria forzada (FEF), con el efecto obstructivo en la vía distal, tal y como ocurre en los pacientes fumadores activos (59).…”

Section: Gravedad De Las Exacerbacionesunclassified

“…Existen investigaciones que afirman que la exposición al polvo ambiental y humos en países en vías de desarrollo o desarrollados (54)(55)(56)(58)(59)(60)(61)(62)(63)(64)68), como el ozono, las partículas en suspensión, el monóxido de carbono (CO), el dióxido de sulfuro (SO 2 ) , el dióxido de nitrógeno (NO 2 ), otros gases (54), materiales industriales, partículas procedentes de la minería y la combustión, productos La educación terapéutica en los pacientes con EPOC químicos agrícolas, el humo de los cigarros presentes en la contaminación atmosférica, asociados al tráfico rodado, es un factor de predisposición a las exacerbaciones (69). Sin embargo existe controversia en cuanto a su relación como factor de causa directa de la enfermedad (54).…”

Section:  Contaminación Atmosféricaunclassified

La educación terapéutica en los pacientes con EPOC

Ayora¹

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La educación terapéutica en los pacientes con EPOCTESIS AgradecimientosPor fin ha llegado el momento. Después de tantos meses en los que había que saltar las barreras que se iban sumando para la realización del proyecto, las mismas , barreras me han hecho conocer personas que lo han hecho posible. Así que es difícil empezar los agradecimientos de esta tesis, ya que son muchas las personas que tiene un trocito de cada una de las páginas que la componen.Voy a empezar agradecer en las primeras personas que confiaron en mí para llevarla a cabo, Isabel y Loreto. Intentando motivarme cuando la captación no era buena y orientándome con sus conocimientos en cada uno de los apartados. Así que solo puedo decir gracias.A mis compañeros de la UJI por compartir y escuchar en mis momentos de agobio y desesperación. Pero sin duda a mi compañera, Vicky, que sin conocerme decido ayudarme en todo lo necesario, por qué sin ti esto no hubiera sido posible, he podido dejar pocos o muchos conocimientos a la ciencia pero sin duda, me llevo una amiga, gracias Vicky.Como no, a todos los pacientes que han, confiado en mí, pensando que yo les estaba aportando mucho, pero eran ellos los que me han enseñado todo lo que se de esta enfermedad. Así que siempre os estaré agradecida.Y a otros que aunque no se vean de forma física han aportado grandes granitos como mi novio y compañero de viaje en el camino de la vida, Carlos, por estar ahí cuando te he necesitado y siempre me has sabido comprender, muchas gracias, por ser tan bueno conmigo.A todos mis amigos, en especial a Raquel e Idoya, que aunque los he dejado un poco de lado los últimos meses siempre han estado motivando, como lo prometido es deuda por fin podré irme al viaje que me regalasteis para mi cumpleaños. A mis padres y hermana, solo me queda decirlos que prometo estar más tiempo con vosotros, ya no me volverá a quedar la excusa de que tengo que hacer tesis, a ti Belén prometo visitarte en Barcelona que hace un año que te fuiste y no he ido ni a ver tu casa, perdón.Y a tantas personas que no recuerdo en este momento pero sin duda tienen mucho que ver con estas páginas. Ana Folch Ayora RESUMENIntroducción: la EPOC es prevenible y tratable. En el curso de la misma aparecen uno o dos periodos de agudizaciones o exacerbaciones anuales, suponiendo un elevado coste sanitario. Por lo tanto la prevención de las exacerbaciones es un objetivo prioritario recogido en las principales guías de consenso. La educación terapéutica (ET) forma parte del tratamiento en la prevención secundaria y terciaria.Objetivo: Determinar y evaluar la efectividad de un programa estructurado de ET, en comparación con la atención tradicional, trascurridos tres meses del alta, en base al uso de recursos sanitarios utilizados, calidad de vida, conocimiento de la enfermedad y la adherencia al tratamiento.Material y métodos: revisión integradora de los ensayos clínicos que apliquen programas de ET, estudio descriptivo de la población EPOC en el Hospital Universitario General de las historias clínicas de los últimos 5 a...

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Passive smoking and respiratory symptoms in the FinEsS Study

Cited by 90 publications

References 28 publications

Socio-economic status is related to incidence of asthma and respiratory symptoms in adults

Socio-economic status is related to incidence of asthma and respiratory symptoms in adults

Sex differences in respiratory symptoms: Fig. 1.—

La educación terapéutica en los pacientes con EPOC

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