1988
DOI: 10.1056/nejm198803033180906
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Pathogenesis and Treatment of Polycystic Ovary Syndrome

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Cited by 175 publications
(70 citation statements)
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“…The frequencies of selfreported polycystic ovaries/SLS (1.35%) and infertility known, for example, if and to what degree the condition is permanent or transient. 4,6 The authors are unamong SLS individuals (27.5%) in this study are similar to those reported among the population-based conaware of any studies that have directly tried to determine the prevalence of SLS. Previous studies using trols of the CASH study 18 : 0.94% reported physiciandiagnosed polycystic ovaries or SLS, and 27.2% of ultrasound to address the question of population prevalence of polycystic ovary morphology (a component those with SLS reported a history of infertility (defined as ''unsuccessful attempts to get pregnant for two or of SLS, but not exclusive to or definitive for the disorder), 21 have suffered from either low response more years'').…”
Section: / 7b4b$$0852supporting
confidence: 82%
“…The frequencies of selfreported polycystic ovaries/SLS (1.35%) and infertility known, for example, if and to what degree the condition is permanent or transient. 4,6 The authors are unamong SLS individuals (27.5%) in this study are similar to those reported among the population-based conaware of any studies that have directly tried to determine the prevalence of SLS. Previous studies using trols of the CASH study 18 : 0.94% reported physiciandiagnosed polycystic ovaries or SLS, and 27.2% of ultrasound to address the question of population prevalence of polycystic ovary morphology (a component those with SLS reported a history of infertility (defined as ''unsuccessful attempts to get pregnant for two or of SLS, but not exclusive to or definitive for the disorder), 21 have suffered from either low response more years'').…”
Section: / 7b4b$$0852supporting
confidence: 82%
“…They are at increased risk of developing PCOS, a group of clinical and biological manifestations involving hyperandrogenism, menstrual disturbances, and chronic anovulation. Although the aetiology of PCOS has not been elucidated (44,45), the condition is reminiscent of that in rodents with leptin deficiency or leptin resistance (46). Studies of leptin concentrations in obese women with PCOS at about the age of 30 years of age failed to reveal differences from normally cyclic obese controls (32,(47)(48)(49)(50)(51).…”
Section: Discussionmentioning
confidence: 99%
“…It can be explained due to abnormality of the hypothalamic-pituitary ovarian or adrenal axis has been implicated in PCOS. Disturbance in gonadotrophin releasing hormone (GnRH) results in the relative increase in LH to FSH release, abnormal feedback mechanism by ovarian estrogen is blamed to play role in this discriminated increase in LH release (25) . Serum zinc level was significantly decreased in obese PCO group than in obese control group with P value < 0.02, which is different from other study which showed no significant difference between patients group and control group with respect to serum zinc levels (P > 0.05) (26) .…”
Section: Discussionmentioning
confidence: 99%