Persistence of obstructive sleep apnea syndrome in children after adenotonsillectomy

Tauman, Riva; Gulliver, Tanya; Krishna, Jyoti; Montgomery‐Downs, Hawley E.; O’Brien, Louise M.; Ivanenko, Anna; Gozal, David

doi:10.1016/j.jpeds.2006.08.067

Cited by 397 publications

(282 citation statements)

References 27 publications

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“…Polysomnographic findings improved markedly after adenotonsillectomy, with more children in the earlyadenotonsillectomy group than in the watchful-waiting group having normalization of polysomnographic findings (79% vs. 46%). Normalization of the polysomnographic findings in the large majority of children randomly assigned to early adenotonsillectomy is consistent with some, 34,35 but not all, 36,37 studies. Previous nonrandomized studies may have been limited by differential follow-up of more symptomatic children.…”

Section: Discussionsupporting

confidence: 83%

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

Marcus

Moore

Rosen

et al. 2014

Survey of Anesthesiology

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229

438

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BACKGROUND-Adenotonsillectomy is commonly performed in children with the obstructive sleep apnea syndrome, yet its usefulness in reducing symptoms and improving cognition, behavior, quality of life, and polysomnographic findings has not been rigorously evaluated. We hypothesized that, in children with the obstructive sleep apnea syndrome without prolonged oxyhemoglobin desaturation, early adenotonsillectomy, as compared with watchful waiting with supportive care, would result in improved outcomes.

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Section: Discussionsupporting

confidence: 83%

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

Marcus

Moore

Rosen

et al. 2014

Survey of Anesthesiology

Self Cite

229

438

View full text Add to dashboard Cite

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“…But the first line of treatment for Review many children with OSA is still AT. Several studies, however, have shown that many children subjected to AT surgery present residual SDB [3,4,5,7,29,30,31,32]. The addition of orthodontic treatment in these children may help to further reduce the residual AHI, but many questions remain unresolved, i.e., role of orthodontics in the treatment of OSA and its placement compared to adenotonsillectomy.…”

Section: Discussionmentioning

confidence: 99%

Rapid maxillary expansion before and after adenotonsillectomy in children with obstructive sleep apnea

2012

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In children adenotonsillectomy (AT)\ud is the recommended treatment in the\ud presence of obstructive sleep apnea\ud (OSA) [1]. It should be performed, if\ud needed, in association with nasal inferior\ud turbinate reduction usually\ud with radiofrequency [2]. But as shown\ud in previous reports, upper airway soft\ud tissues treatment is not always successful\ud in completely controlling abnormal\ud breathing during sleep in children,\ud despite the fact that subjective\ud improvement is often reported [3, 4,\ud 5, 6]

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“…First, many studies have shown that the use of T&A in pediatric OSA patients may have variable results, reaching an AHI of 1 or less in about 50% of cases (and as low as 32% in obese children) [23][24][25][26][27] . However, a long-term follow-up study [28] performed first in 6-to 12-year-old children with OSA and repeated in 4-to 6-year-old children with OSA who underwent T&A showed progressive recurrence and worsening in both "apparently cured" and "significantly improved" children, and in 68% of the children after 36 months of follow-up in the first study.…”

Section: Negative Effect Of Not Addressing the Dysfunctionmentioning

confidence: 99%

Pediatric Obstructive Sleep Apnea: Where Do We Stand?

Huang

Guilleminault

2017

Advances in Oto-Rhino-Laryngology

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Pediatric obstructive sleep apnea (OSA) was initially described in 1976. In 1981, Dr. Guilleminault emphasized that pediatric OSA was different from the clinical presentation reported in adults. It was characterized by more disturbed nocturnal sleep than excessive daytime sleepiness, and presented more behavioral problems, particularly school problems, hyperactivity, nocturnal enuresis, sleep terrors, depression, insomnia, and psychiatric problems. The underlying causes of pediatric OSA are complex. Such factors as adenotonsillar hypertrophy, obesity, anatomical and neuromuscular factors, and hypotonic neuromuscular disease are also involved. Adenotonsillectomy (T&A) has been the recommended treatment for pediatric OSA, but in the recent past this practice has been placed very much in question. Therefore, we will discuss the mechanism of pediatric OSA and investigate obese and nonobese pediatric sleep-disordered breathing. Moreover, the important concept that dysfunction leads to the dysmorphism that impacts on the size of the Lin H-C (ed): Sleep-Related Breathing Disorders.

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Persistence of obstructive sleep apnea syndrome in children after adenotonsillectomy

Cited by 397 publications

References 27 publications

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

Rapid maxillary expansion before and after adenotonsillectomy in children with obstructive sleep apnea

Pediatric Obstructive Sleep Apnea: Where Do We Stand?

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