Physiological cardiac pacing: an individual objective

Daubert, J.-C.; Mabo, Philippe; Gras, Daniel; Leclercq, Christophe; Lelièvre, T

doi:10.1007/978-94-011-0872-0_23

Cited by 7 publications

(15 citation statements)

References 51 publications

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“…However, excessive lengthening the delay in patients with atrioventricular block can paradoxically lead to the fall of CO, which by itself may be more detrimental than the right ventricular pacing. 8 Patients with second and third degree atrioventricular block are inevitably destined to depend on the right ventricular pacing. 18,19 When AVD is programmed optimally, it may improve CO by 13-40%.…”

Section: Discussionmentioning

confidence: 99%

“…3,4 Although longterm structural remodeling and dyssynchrony may be avoided with optimal right ventricular pacing site, correct programming of atrioventricular delay (AVD) may offer additional hemodynamic benefits. 8 The problem of the individual AVD optimization was noticed already in the 1980s. 5,6 Programming of AVD influences contractile function of left ventricle through preload modulation, 7 and if done incorrectly, can have unfavorable hemodynamic results.…”

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confidence: 99%

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Influence of Atrioventricular Optimization on Hemodynamic Parameters and Quality of Life in Patients with Dual Chamber Pacemaker with Ventricular Lead in Right Ventricular Outflow Tract

Klimczak

Budzikowski

Rosiak

et al. 2014

Noninvasive Electrocardiol

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Section: Discussionmentioning

confidence: 99%

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confidence: 99%

Influence of Atrioventricular Optimization on Hemodynamic Parameters and Quality of Life in Patients with Dual Chamber Pacemaker with Ventricular Lead in Right Ventricular Outflow Tract

Klimczak

Budzikowski

Rosiak

et al. 2014

Noninvasive Electrocardiol

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“…However, this maneuver lengthens the total atrial refractory period of the pacemaker and lowers the 2:1 point to the low level of 100–120 beats/min, thereby restricting exercise tolerance. This solution is acceptable only in elderly and inactive patients 167 Biatrial synchronous DDD pacing.…”

Section: Hemodynamic Consequences Of Intra‐ and Interatrial Conductiomentioning

confidence: 99%

Intra‐ and Interatrial Conduction Delay:

Daubert

Pavin

Jauvert

et al. 2004

Pacing Clinical Electrophis

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Atrial conduction disorders are frequent in elderly subjects and/or those with structural heart diseases, mainly mitral valve disease, hyperthrophic cardiomyopathies, and hypertension. The resultant electrophysiological and electromechanical abnormalities are associated with a higher risk of paroxysmal or persistent atrial tachyarrhythmias, either atrial fibrillation, typical or atypical flutter or other forms of atrial tachycardias. Such an association is not fortuitous because intra- and interatrial conduction abnormalities delays disrupt (spatial and temporal dispersion) electrical activation, thus promoting the initiation and perpetuation of reentrant circuits. Preventive therapeutic interventions induce variable, sometimes paradoxical effects as with the proarrhythmic effect of class I antiarrhythmic drugs. Similarly, atrial pacing may promote proarrhythmias or an antiarrhythmic effect according to the pacing site(s) and mode. Multisite atrial pacing was conceived to correct, as much as possible, abnormal activation induced by spontaneous intra- or interatrial conduction disorders or by single site atrial pacing, which are situations responsible for commonly refractory arrhythmias. Atrial electrical resynchronization can also be used to correct mechanical abnormalities like left heart AV dyssynchrony resulting from intraatrial conduction delays.

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“…Atrio-ventricular delay (AVD) during dual-chamber pacing influences left ventricle contraction function through preload modulation [5]. Improperly programmed AVD in the DDD pacemaker can have unfavorable hemodynamic results [6].The aim of this manuscript is to review the clinical and technical aspects of AVD optimization and existing methods to define the optimal AVD in patients with DDD pacemaker. …”

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confidence: 99%

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confidence: 99%