2019
DOI: 10.1016/j.ekir.2019.01.010
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Pigment Nephropathy in a Patient With PYGM Gene Mutation

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“…Due to this direct vasoconstrictive effect of calcium the renal consequences that occur are: arteriolar vasoconstriction, reduction in glomerular ultrafiltration coefficient (Kf), reduction in tubular sodium reabsorption, nephrogenic diabetes insipidus, prerenal azotaemia, acute tubular necrosis, nephrocalcinosis, and tubulointerstitial fibrosis [11]. This intrarenal vasoconstriction is also one of the mechanism of renal toxicity in rhabdomyolysis [12]. In the present case both prerenal involvement or direct vasoconstrictive effect of the calcium ion on vascular smooth muscle can be the probable mechanism of AKI secondary to hypercalcemia.…”
Section: Discussionmentioning
confidence: 99%
“…Due to this direct vasoconstrictive effect of calcium the renal consequences that occur are: arteriolar vasoconstriction, reduction in glomerular ultrafiltration coefficient (Kf), reduction in tubular sodium reabsorption, nephrogenic diabetes insipidus, prerenal azotaemia, acute tubular necrosis, nephrocalcinosis, and tubulointerstitial fibrosis [11]. This intrarenal vasoconstriction is also one of the mechanism of renal toxicity in rhabdomyolysis [12]. In the present case both prerenal involvement or direct vasoconstrictive effect of the calcium ion on vascular smooth muscle can be the probable mechanism of AKI secondary to hypercalcemia.…”
Section: Discussionmentioning
confidence: 99%