Pityriasis lichenoides and its subtypes

Mucha-Habermann Disease-like Eruption Following Pandemic Influenza H1N1 Vaccination: Coincidence or Causal Relationship?

“…of the extremities. MHD is speculated to be an inflammatory reaction triggered by certain infectious agents, an inflammatory response secondary to T-cell dyscrasia, or an immune complex-mediated hypersensitivity [2]. Various pathogens have been proposed to cause MHD, such as Epstein-Barr virus, Toxoplasma gondii, adenovirus, Parvovirus B19, Staphylococcus aureus and Streptococcus pyogenes.…”

Asymptomatic, Reddish Papules and Pustules with Hemorrhagic Crust on the Trunk of a Girl: Answer of the Last Issue’s Case Question

“…1,2 Although descriptions of this disorder had been made many times previously, term "PLEVA" was first described in 1925 by Habermann. 3 PLEVA occurs predominantly in the second or third decades of life and it is uncommon in childhood. There is a male predominance.…”

“…4 The disease starts on the trunk with erythematous papules that develop crusts, vesicles, pustules or erosions, then spontaneously regresses within a few weeks. 3,4 Varioliform scars and postinflammatory hyper-or hypopigmentation may result. 3 The eruption may be asymptomatic or sometimes accompanied by itching, fever and malaise.…”

“…3,4 Varioliform scars and postinflammatory hyper-or hypopigmentation may result. 3 The eruption may be asymptomatic or sometimes accompanied by itching, fever and malaise. 1 There are three major pathogenic theories of PLEVA: an inflammatory reaction triggered by infectious agents; an inflammatory response secondary to a T-cell dyscrasia and an immune complex-mediated hypersensitivity vasculitis.…”

“…1 There are three major pathogenic theories of PLEVA: an inflammatory reaction triggered by infectious agents; an inflammatory response secondary to a T-cell dyscrasia and an immune complex-mediated hypersensitivity vasculitis. 3 A number of physicians suspect an infectious etiology such as Epstein-Barr virus, Toxoplasma gondii and Human immunodeficiency virus since the disease appears rapidly and favors younger patients. 2,3 Several reports have demonstrated clonal T-cell receptor rearrangements from patient specimens, suggesting that PLEVA is a lymphoproliferative disorder involving T-cell subtypes, despite its clinically benign course and the absence of morphologically atypical cells in the skin lesions.…”