Platypnea-Orthodeoxia Syndrome Associated with Spontaneously Ruptured Chordae Tendineae of Tricuspid Valve

Uchihashi, Motoki; Makino, Masahiro; Kaimoto, Satoshi; Imai, Y; Hadase, Mitsuyoshi; Kurata, Hiroyuki; Nakamura, Takashi

doi:10.1016/j.case.2019.10.006

Cited by 2 publications

(2 citation statements)

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“…Both TTE and TEE were performed in our patient, enabling us to measure the diameter of the PFO, which was 4 mm. Diameters of up to 6.3 mm or 15 mm have been reported in the literature [17] [18].…”

Section: In the Study Bymentioning

confidence: 96%

Platypnea-Orthodeoxia Syndrome in an Elderly Patient Treated with Percutaneous Patent Foramen Ovale Closure: Report of a Case and Review of the Literature

Diouldé,

Mariama,

Madiou

et al. 2023

WJCD

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Introduction: Platypnea-Orthodeoxia syndrome is characterized by dyspnea and hypoxia in the upright position, usually improving in the supine position. Two components are required: an interatrial or intrapulmonary shunt, and a functional component. Diagnosis is made by contrast ultrasonography. We report a case of Platypnea-Orthodeoxia syndrome revealed by positional dyspnea in an 87-year-old patient. The aim of this study is to describe the clinical, therapeutic and evolutionary profile of this syndrome. Case Presentation: This is an 87-year-old patient with a history of pulmonary embolism (PE) and stroke. He was seen for dyspnea and desaturation in orthostatism, revealing a patent foramen ovale (PFO). Progression was favorable after closure of the PFO. Conclusion: Platypnea-Orthodeoxia syndrome may be presented as simple exertional dyspnea. The clinician should check for improvement in symptoms and/or oxygenation during decubitus. Definitive treatment consists of percutaneous closure of the shunt.

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Section: In the Study Bymentioning

confidence: 96%

Platypnea-Orthodeoxia Syndrome in an Elderly Patient Treated with Percutaneous Patent Foramen Ovale Closure: Report of a Case and Review of the Literature

Diouldé,

Mariama,

Madiou

et al. 2023

WJCD

View full text Add to dashboard Cite

show abstract

“…21 Further subsets of anatomic variations that have previously been shown to contribute to POS include a prominent eustachian valve/Chiari network that diverts blood to the superior septum, 22 dilated aortic root or ascending aortic aneurysm that alters the position of the IAS, 23,24 vertebral changes including severe kyphosis, 25 diaphragmatic paralysis, 26 mediastinal shift from thoracic changes including after pneumonectomy, 27 or severe tricuspid regurgitation with jet directed toward IAS. 28,29 Further evidence suggests that anatomic and pressure differences may only explain a component of the shunting. The specific pattern of vortical blood flow in the right and left atria contributes to not only the likelihood of shunting but also the overall maintenance of PFO patency.…”

Section: Platypnea-orthodeoxia Syndromementioning

confidence: 99%