Pleural effusion in HBsAG-positive hepatitis

Cocchi, P; Silenzi, M

doi:10.1016/s0022-3476(76)80503-3

Cited by 5 publications

(2 citation statements)

References 3 publications

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“…In our previous report ( l ) , we suggested that the occurrence of ascites in the course of acute viral hepatitis may be a self-limited, immunopathic sign. Its pathogenesis appears to be similar to that proposed for the "exudative" pleural effusions occasionally associated with acute viral hepatitis B (9)(10)(11)(12). It is possible that both pleural effusions and exudative ascites represent instances of immunocomplex-related manifestations (1 3), although no direct evidence favoring this view could be obtained in the present series.…”

Section: Discussionsupporting

confidence: 65%

Portal pressure in patients with exudative ascites in the course of acute hepatitis B

Calvet

Bruix

Bosch

et al. 1991

Liver

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ABSTRACT— We describe three patients who developed ascites during the course of acute viral hepatitis B. Two of them had exudative ascites, with a high protein and cell content, and the other transudative ascites, with low protein and cell content. Both patients with exudative ascites had a benign clinical course, and their liver disease was milder than in the patient with transudative ascites, who had signs of severe liver failure and a submassive hepatic necrosis on liver biopsy. Moreover, the patient with transudative ascites had evidence of portal hypertension (as indicated by a hepatic vein pressure gradient of 12.5 mmHg, normal 1–6 mmHg), whereas patients with exudative ascites did not (hepatic vein pressure gradient of 5 and 5.5 mmHg, respectively). These data support our previous suggestion that “exudative” ascites during acute viral hepatitis B represents a self‐limited immunopathic sign that is not related to portal hypertension or severe hepatic disease.

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Section: Discussionsupporting

confidence: 65%

Portal pressure in patients with exudative ascites in the course of acute hepatitis B

Calvet

Bruix

Bosch

et al. 1991

Liver

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“…The association of pleural effusion with hepatitis, both HBsAgpositive and HBsAg-negative, has been previously re ported [2] though the mechanism for this association re mains obscure.…”

Section: Introductionmentioning

confidence: 96%

Hepatitis B Virus: A Possible Cause of Serositis in Hemodialysis Patients

Dave¹,

Choi²,

Cohen³

1983

Nephron

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An epidemiologic survey in a maintenance hemodialysis population of 300 patients was undertaken to relate the appearance of acute serositis (pericarditis, pleuritis or ascites) to HBsAg antigenemia. A significant number of incidents of serositis occurred in patients acutely or chronically infected with hepatitis B surface antigen (HBsAg) suggesting an etiologic role for the virus in the serositis of uremia. In 2 patients with both end-stage renal disease and chronic HBsAg antigenemia, immunofluorescent studies of serosal tissues showed fluorescent clusters interpreted to be HBs antigen-antibody complexes. It is concluded that an immunologic response to viremia may be one of the causes of serositis in uremia.

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