Polyarteritis nodosa associated with streptococcus.

David, Julie; Ansell, Barbara M.; Woo, P

doi:10.1136/adc.69.6.685

Cited by 87 publications

(39 citation statements)

References 8 publications

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“…Taken together, patients with FMF certainly have an exaggerated response to streptococcal antigens, and they may be more prone to the late complications of streptococcal infections. Interestingly, FMF patients associated with PAN had higher prevalence of ASO elevation [57]. Besides streptococcal infections, there are also case reports noting the presence of viral infections such as hepatitis A preceding the occurrence of HSP or PFM in FMF patients [58][59][60][61].…”

Section: Pioneering Reports About Fmf-vasculitis Associationmentioning

confidence: 97%

Coexistence of vasculitides with Familial Mediterranean Fever

Aksu

Keser

2011

Rheumatol Int

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Familial Mediterranean fever (FMF) is the most common autoinflammatory disease characterized by recurrent self-limited attacks of fever accompanied with peritonitis, pleuritis, or arthritis. FMF may coexist with various systemic inflammatory diseases including vasculitides, spondyloarthritis, multiple sclerosis, and inflammatory bowel disease. Among these coexistences, this review concentrates on vasculitic disorders, with the aim of increasing the awareness of FMF-vasculitis association. This association does not merely show a coincidentally increased frequency of vasculitic disorders in FMF; rather, it seems that FMF patients might be at increased risk of developing vasculitis. Indeed, as also suggested by some authors, vasculitis might be an essential feature of FMF. Among the vasculitic disorders reported to be associated with FMF, Henoch-Schönlein purpura, and classical polyarteritis nodosa come the first, possibly followed up by protracted febrile myalgia. There is also an ongoing debate whether Behçet's disease (BD) more frequently seen in FMF than expected by chance alone. In this review, the associations of various vasculitic disorders with FMF and the possible pathogenic mechanisms underlying these associations, as well as the frequencies and clinical significances of FMF-related MEFV mutations in various vasculitides including BD, are discussed in the context of the available data.

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Section: Pioneering Reports About Fmf-vasculitis Associationmentioning

confidence: 97%

Coexistence of vasculitides with Familial Mediterranean Fever

Aksu

Keser

2011

Rheumatol Int

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“…1,15 As for ANCA, Pereira et al 16 reported the first detection of serum myeloperoxidase-specific ANCA (p-ANCA) in patient with cutaneous PAN. However, ANCA is usually absent in cutaneous PAN, as shown in the review of David et al 17 of six patients with cutaneous PAN who were tested for ANCA and were all found to be negative. Besides HBV, other infectious agents, such as streptococcus, 1,17,18 Mycobacterium tuberculosis, 19 and human immunodeficiency virus 20,21 have also been implicated in the pathogenesis of cutaneous PAN.…”

Section: Discussionmentioning

confidence: 89%

Manifestation of cutaneous polyarteritis nodosa during interferon therapy for chronic hepatitis C associated with primary biliary cirrhosis

Dohmen¹,

Miyamoto²,

Irie³

et al. 2000

J Gastroenterol

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Interferon alpha-2b was administered to a 50-year-old Japanese woman with chronic hepatitis C associated with primary biliary cirrhosis. Two months after the beginning of the interferon alpha-2b therapy a systemic nodular, erythematous rash developed. Histological analysis of the skin revealed typical features of necrotizing arteritis. Because there was no microhematuria, and no microaneurysms were detected on abdominal angiography, a diagnosis of cutaneous polyarteritis nodosa was made. A good outcome was achieved after interferon alpha-2b was discontinued and prednisolone was administered instead. The cutaneous polyarteritis nodosa in this patient is thus considered to have occurred as an adverse effect of interferon administration. To our knowledge, this is the first reported case of cutaneous polyarteritis nodosa which developed because of interferon therapy for chronic hepatitis C associated with primary biliary cirrhosis.

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“…The fact that the causing organisms of reported cases are S. viridans and A. actinomybetemcomitans, both bacteria of low virulence, might support the hypothesis. Poststreptococcal vasculitis has previously been described in 12 children [10]. These patients had fever, arthralgia or arthritis, myalgia, and vasculitic rashes following streptococcal upper respiratory infection.…”

Section: Discussionmentioning

confidence: 96%

Vasculitis associated with septicemia: case report and review of the literature

et al. 2001

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We report an unusual case in which infectious endocarditis presented systemic vasculitis and glomerulonephritis as the initial manifestation of the disease. The patient was a 16-year-old girl with congenital cyanotic heart disease who presented with skin purpura, proteinuria, and hematuria. She had hypergammaglobulinemia, cryoglobulinemia, and positive circulating immune complexes. Renal biopsy revealed crescentic glomerulonephritis. Her serum C3 level, which was initially normal, became decreased, and prednisolone and azathioprine were administered with a tentative diagnosis of systemic lupus erythematosus (SLE). Soon after, she developed fever and renal failure. Blood culture grew Streptococcus pyogenes, and the diagnosis of infectious endocarditis was made. Eight cases of systemic vasculitis and glomerulonephritis associated with infectious endocarditis have been described in the literature. Infectious endocarditis should be included in the differential diagnosis of systemic vasculitis and glomerulonephritis.

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Polyarteritis nodosa associated with streptococcus.

Cited by 87 publications

References 8 publications

Coexistence of vasculitides with Familial Mediterranean Fever

Coexistence of vasculitides with Familial Mediterranean Fever

Manifestation of cutaneous polyarteritis nodosa during interferon therapy for chronic hepatitis C associated with primary biliary cirrhosis

Vasculitis associated with septicemia: case report and review of the literature

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