Post-COVID-19 vaccine Guillain-Barré syndrome; first reported case from Qatar.

Abstract

Guillain-Barré syndrome is an immune-mediated neuropathy that was reported following multiple vaccines. We present the case of a gentleman who developed GBS 20 days after the second dose of COVID-19 vaccination. It is important to mention that more research is needed to establish an association between COVID-19 vaccine and GBS.

Causality or coincidental: The first case report of acute inflammatory demyelinating polyneuropathy following COVID-19 vaccination in the Philippines

“…Because of the limited case reports, no guidelines on the optimal treatment of vaccine-associated GBS are available. Most cases have been treated with IVIG (2 g/kg over 3-5 days) [3,[5][6][7][8][9][10].…”

“…Indeed, there have been case reports of GBS attributed to COVID-19 vaccines (Johnson and Johnson, Pfizer, AstraZeneca) that have been published in United States, United Kingdom, Israel, Egypt, Austria and Qatar. Based on these published case reports, the onset from the time of vaccination to the development of neuropathic symptoms can be as short as one day to as much as twenty-one days [3][4][5][6][7][8][9]1]. To date, there have been no published case reports in the Philippines regarding COVID-19 vaccine and the development of neuropathic symptoms attributed to AIDP.…”

Guillain‐Barré syndrome following COVID‐19 vaccination: An updated systematic review of cases

“…Studies describing the cases of Guillain Barré Syndrome following COVID-19 vaccination (Figure 1); in addition, the analysis did not include review articles or consensus statements. The Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) were used to illustrate inclusions and exclusions [20]. We discovered 113 studies from PubMed, Google Scholar, Ovid, Web of Science, and Scopus based on our search parameters.…”

Síndrome De Guillain–barré E Vacinação Contra a Covid-19: Uma Revisão De Literatura

“…Nas neuropatias axonais agudas, não costuma haver desmielinização, e as lesões incluem apenas axônios dos nervos motores e sensoriais (Principi & Esposito, 2019). A ocorrência da síndrome após infecções reforça a teoria do mimetismo molecular, que postulou que alguns patógenos, como por exemplo o Campylobacter jejuni, podem levar à formação de anticorpos de reação cruzada, que visam gangliosídeos que constituem uma parte da bainha de mielina que circunda os nervos periféricos (RAZOK,, et al 2021).…”