Posterior fossa epidermoid cysts

Intracranial epidermoid tumors are rare, potentially curable, benign lesions that are sometimes associated with perioperative complications, and tend to recur if not completely removed. Histologically benign epidermoid tumors may also develop into highly malignant tumors. This study evaluated on 28 cases of intracranial epidermoid tumor treated over a 13-year period by radical resection with microneurosurgical techniques. The majority of patients underwent computed tomography and/or magnetic resonance imaging within the first 24 hours postoperatively to confirm the results of surgery. Radical surgical resection was achieved in 21 of the 28 cases, and there was no operative mortality. The most common postoperative complication was transient paresis of various cranial nerves. During a mean follow up of 6 years, only one tumor became malignant. Radical surgical resection should be the goal in treating these benign lesions, but if not possible, every effort should be made to minimize the amount of tumor tissue that remains.

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Surgical Treatment of Intracranial Epidermoid Tumors

Akar

Tanrıöver

Tüzgen

et al. 2003

“…Epidermoids of the brain are rare tumors, accounting for approximately 1% of all brain tumors, [1][2][3]13,22) and are usually located in the cerebellopontine angle 1,5,10,14,15,17,18) and parasellar region. 1,21) Epidermoid tumors of the fourth ventricle account for between 5% and 31.4% of all intracranial epidermoid tumors.…”

Section: Introductionmentioning

confidence: 99%

Reversible Bilateral Vestibular Impairment Caused by Fourth Ventricle Epidermoid Tumor-Case Report-

Kambe

Anno

Oda

et al. 2003

A 26-year-old man presented with an epidermoid tumor of the fourth ventricle manifesting as headache with nausea and vertigo. Neurological examination revealed no cerebellar signs, except nystagmus. Bilateral vestibular impairment was identified by the caloric test. The tumor was removed via the midline suboccipital approach. The bilateral peripheral vestibular function recovered remarkably postoperatively. This marked improvement suggests that the bilateral vestibular impairment was caused by compression of the vestibular nuclei by the tumor.

“…2,5,6) Nevertheless, we have identified 10 cases of intracranial epidermoid tumor with intratumoral hemorrhage. 1,[4][5][6][7]9,10,12,14,16) Although the CT finding of slight hyperdensity suggested the presence of blood products or calcification, no histological evidence of hemorrhage was found in our case.…”

Section: Discussionmentioning

confidence: 71%

“…6) The cerebellopontine angle is the most common site of occurrence of approximately 40% of intracranial epidermoid tumors, 4) which account for 5% of the tumors in this region. 12) The tumor is cystic and the walls are formed of keratinizing stratified squamous epithelium without skin appendages. Most epidermoid tumors appear as hypointense on T 1 -weighted magnetic resonance (MR) images and hyperintense on T 2 -weighted MR images.…”

Section: Introductionmentioning

confidence: 99%

Intracranial Epidermoid Tumor With Changes in Signal Intensity on Magnetic Resonance Imaging Because of Non-hemorrhagic Pathology -Case Report-

Muto

Yoshida

Momoshima

et al. 2010

A 61-year-old woman presented an intracranial epidermoid tumor manifesting as dizziness and right facial hypesthesia. Magnetic resonance (MR) imaging revealed a well-defined lobulated mass in the right cerebellopontine angle as nearly isointense to the cerebrospinal fluid (CSF) on both T 1 -and T 2 -weighted images but inhomogeneously hyperintense on fluid-attenuated inversion recovery images. MR imaging performed 1 year later revealed that the tumor had significantly enlarged, and now appeared hyperintense to the CSF on T 1 -and T 2 -weighted images. The lesion was confirmed at surgery to be an epidermoid tumor filled with xanthochromic fluid. Histological examination found no evidence of hemorrhage in the resected tumor, so the changes in the MR imaging signal intensity were attributed to changes in the protein concentration of the intratumoral fluid, accumulation of debris, or some other non-hemorrhagic process.