Posterior Lenticonus

Abstract

Two patients with posterior lenticonus; one was unilateral and the other was bilateral. The second patient had bilateral cataract extractions and the lenses were examined histopathologically. Postoperatively she did not have any amblyopia. The purpose of this presentation is to illustrate two points from the clinicopathological findings: The pathogenesis of the anomaly is mainly due to inherited thinning and weakness of the central part of the posterior lens capsule. We suggest that the posterior lenticonus without complicated cataract is not a main cause of profound amblyopia. The amblyopia is dependent on the age at which the cataract develops.

Persistent central posterior capsule bulging after cataract extraction for posterior lenticonus

“…4 Cause of lenticonus is considered to be the thinning and bending of the posterior capsule that leads to progressive distortion of the lens fibers and cataract formation. 2,5 In our case it seems that even after cataract extraction, the posterior capsule still preserved its preoperative configuration with a central well-defined bulging part. Surgeons should be informed about this post cataract finding as a sign of pre-existing posterior lenticonus (frequently accompanied by postoperative amblyopia) and distinguish it from other clinical entities with similar clinical findings (such as, postoperative capsular block syndrome).…”

Intraoperative posterior capsule flutter in posterior lentiglobus

“…[1][2][3] The pathogenesis of posterior lentiglobus involves subepithelial capsular hyperplasia, embryologic hyaloid artery traction, inherent weakness of the posterior capsule wall, and excessive strain during accommodation, resulting in posterior herniation of the lens. 4 We previously hypothesized 5 that posterior lentiglobus might be a prerequisite for a posterior capsule defect.…”

Morning Glory Syndrome Associated with Posterior Lenticonus

“…Khalil and Saheb [13] report ed that posterior lenticonus is most likely caused by herniation of cortical lens fibers and posterior capsule into the vitreous at an area of posterior capsule weakness during fetal development. Franceschetti and Rickli [14] postulated that the anomaly is an aberrant hyperplasia of the subcapsular epithelium overlying the cone.…”