Posterior vitreous detachment following panretinal laser photocoagulation

Sebag, J.; Buzney, Sheldon M.; Belyea, David A.; Kado, Masanori; McMeel, J. Wallace; Trempe, Clement L.

doi:10.1007/bf02764282

Cited by 40 publications

(20 citation statements)

References 21 publications

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“…Sebag and Nguyen-Cuu [30] reported that the risk of PDR is lower in eyes with PVD than in eyes with an attached vitreous, a well-established finding [31, 32]. It was hypothesized previously that PRP might provide therapeutic benefit by inducing a PVD, and studies have shown that the incidence of PVD was greater in patients treated with PRP than in those who did not, supporting this hypothesis.…”

Section: Discussionmentioning

confidence: 51%

Comparison of 577-nm Multispot and Standard Single-Spot Photocoagulation for Diabetic Retinopathy

et al. 2018

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Objective: To compare two different laser strategies of panretinal photocoagulation for diabetic retinopathy. Methods: Single-center, randomized study including 41 eyes treated with 577-nm multispot laser with a 20-ms pulse duration (group 1) or a 532-nm single-spot laser with a 100-ms pulse duration (group 2). The outcomes included best-corrected visual acuity (BCVA) and imaging changes at baseline, 6 and 12 months, laser parameters, and results of subjective pain analysis. Results: At 12 months, the treatments did not differ significantly in BCVA, central retinal thicknesses (CRTs), improved macular edema, vitreomacular interface changes, patient-reported pain scores, or angiographic responses. Group 1 had significantly fewer treatment sessions but used more laser spots (p < 0.001). Conclusion: The multispot laser required fewer applications with more spots delivered to compensate for lower fluency, showing similar patient tolerance to single-spot laser. Both groups maintained the initial visual acuities and CRTs; about 50% of cases had vitreomacular interface changes and improved macular edema, with similar angiographic improvements after 12 months.

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Section: Discussionmentioning

confidence: 51%

Comparison of 577-nm Multispot and Standard Single-Spot Photocoagulation for Diabetic Retinopathy

et al. 2018

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“…Panretinal laser argon photocoagulation therapy has been shown to induce a total PVD over time 19. There is conflicting evidence on whether anti-VEGF injections induces changes in VRI status; a prospective observational study by Geck et al 20 including 61 eyes with a variety of retinal diseases showed development of a PVD in 15 out of 61 eyes (24.5%) while a cohort study by Veloso et al 21 investigating the development of a PVD in 396 eyes with age-related macular degeneration treated with anti-VEGF showed development of a PVD rarely (5.6% of eyes after an average of 8.3 injections).…”

Section: Discussionmentioning

confidence: 99%

A retrospective study of the influence of the vitreomacular interface on macular oedema secondary to retinal vein occlusion

et al. 2017

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“…Previous clinical findings indicated that PDVR is rare if the vitreous cortex has detached completely in myopics with PVD and after vitrectomy, since the scaffold for proliferating cells is destroyed [97]. In high myopia lesser diabetic involvements of the fundus oculi have been observed [57], which may serve as a proof for the involvement of the vitreous in the development of diabetic retinal changes and therefore justifies the term PDVR.…”

Section: Hypothetical Pathogenesis Of Pdvrmentioning

confidence: 99%

Pathogenesis and Classification of Proliferative Diabetic Vitreoretinopathy

Kroll¹,

Rodrigues²,

Hoerle³

2007

Ophthalmologica

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Purpose: To review the current knowledge regarding the pathogenesis of proliferative diabetic vitreoretinopathy (PDVR) and to present recommendations for its clinical staging. Design: Focused literature review and authors’ clinical experience. Results: Although several biochemical mediators may be responsible for the pathogenesis of PDVR, no common biochemical pathway exists. Of those mediators, vascular endothelial growth factor is the one most studied so far. However, since in proliferative diabetic retinopathy (PDR) the thickened posterior vitreous cortex is one of the main factors in the development of proliferations, a consequent shrinkage of the posterior vitreous cortex leads to hemorrhages and tractive retinal detachments. Therefore, PDR should be called PDVR. In consequence, the authors present a new morphological classification of PDVR. Conclusions: There is no consensus about the biochemical pathway responsible for the progression of PDVR. Although several classifications are described in the literature, the classification suggested here is important in the judgment of, the communication about and the therapy of diabetic retinopathy. Furthermore, it is the only reliable classification for predicting the surgical outcome in diabetics.

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Posterior vitreous detachment following panretinal laser photocoagulation

Cited by 40 publications

References 21 publications

Comparison of 577-nm Multispot and Standard Single-Spot Photocoagulation for Diabetic Retinopathy

Comparison of 577-nm Multispot and Standard Single-Spot Photocoagulation for Diabetic Retinopathy

A retrospective study of the influence of the vitreomacular interface on macular oedema secondary to retinal vein occlusion

Pathogenesis and Classification of Proliferative Diabetic Vitreoretinopathy

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