Predictors of Early, Late, and Very Late Stent Thrombosis After Primary Percutaneous Coronary Intervention With Bare-Metal and Drug-Eluting Stents for ST-Segment Elevation Myocardial Infarction

Brodie, Bruce R.; Pokharel, Yashashwi; Garg, Ankit; Kissling, Grace E.; Hansen, C.; Milks, Sally; Cooper, Michael J.; McAlhany, Christopher; Stuckey, Tom

doi:10.1016/j.jcin.2012.06.013

Cited by 61 publications

(41 citation statements)

References 23 publications

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“…We demonstrated that ACS, longer stent length, and lack of CCB use were associated with an increased risk of ST after DES implantation within 1 year in [5]. If subjects were limited to those with ACS who underwent PCI, this tendency was more prominent [14]. Therefore, there are limited data to predict ST occurrence in Japan.…”

Section: Discussionmentioning

confidence: 96%

Relation between stent thrombosis and calcium channel blocker after drug-eluting stent implantation

Nagano

Hokimoto

Nakao

et al. 2015

Journal of Cardiology

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Section: Discussionmentioning

confidence: 96%

Relation between stent thrombosis and calcium channel blocker after drug-eluting stent implantation

Nagano

Hokimoto

Nakao

et al. 2015

Journal of Cardiology

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“…No significant difference was observed between BMS and DES in terms of the incidence of death and recurrent AMI [1,2,6,[10][11][12]. On the other hand, accumulating data began to show a higher risk of stent thrombosis in those treated with DES over a 1-year follow-up period [3,6,9]. Kalesan et al [8] demonstrated in meta-analysis that 1 year after AMI treatment, very late SO is significantly more likely to occur in patients receiving DES than in those with BMS placement.…”

Section: Discussionmentioning

confidence: 99%

Incidence and Clinical Predictors of Stent Restenosis and Early Stent Occlusion in Patients with Acute Myocardial Infarction Treated by Bare Metal Stents: Importance of Infarct Location and Serum Creatinine Level

Uemori¹,

Sugitani²,

Tamada³

et al. 2014

Angiol

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GroupsAmong these 242 patients, 226 underwent either ischemia-driven or planned follow-up coronary angiography within 8 months. Thus, *Corresponding author: Manabu Shirotani, Cardiology Department, Nara Hospital, Kinki University Faculty of Medicine, 1248-1, Otoda-Cho, Ikoma, Nara, 630-0293, Japan, Tel: +81 743 77 0880; Fax: +81 743 77 0901; E-mail: manabu@nara.med.kindai.ac.jp AbstractBackground: Bare Metal Stents (BMS) have been commonly used for recanalization of an infarct-related artery in Japanese patients with Acute Myocardial Infarction (AMI). We sought to examine predictors of binary restenosis and early Stent Occlusion (SO) in these patients. Methods:Among 242 consecutive patients with AMI treated by BMS implantation as reperfusion therapy, 226 underwent either ischemia-driven or follow-up coronary angiography within 8 months. Restenosis change in the stented segment was found in 56. Among them, 10 patients had early SO on an angiogram. Multivariate analysis was performed to obtain predictors of restenosis and early SO.Results: Predictors for restenosis were Left Anterior Descending Artery (LAD) involvement (odds ratio (OR) 2.32, p=0.024), serum creatinine (SCr) on admission (OR 1.29 per 0.1mg/dl increase, p=0.001), and stent size (OR 0.43 per 0.5mm increase, p=0.001). Those for early SO were left main trunk or LAD involvement (OR 27.0, p=0.029), SCr (OR 1.65 per 0.1mg/dl increase, p=0.005) and leukocyte count (OR 1.28 per 1,000/microliter increase, p=0.037) on admission. SCr was significantly higher in patients with early SO than in those with restenosis (median 1.05, Interquartile Range (IQR) 0.80-1.10 vs. median 0.80, IQR 0.70-1.00, p=0.035). Conclusion:In patients with AMI treated with BMS, both restenosis and early SO were increased by anterior wall involvement and elevation of SCr level. Higher SCr may be subject to more occlusive changes. It is suggested that in early SO, an inflammatory mechanism may be involved.

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“…Retrospective registries with intravascular ultrasound (IVUS) imaging in patients with BMS-ST, report of stent underexpansion as the cause of early ST in more than every second case (18). Indeed, severe stent underexpansion is more common in subjects with early ST, suggesting that early and late ST have different underlying mechanisms (4,7,19). Underexpansion and acute malapposition occurs as a result of inadequate stent expansion during index PCI.…”

Section: Uncovered Struts Underexpansion Malapposition and Neoathermentioning

confidence: 99%

Malapposed, uncovered, underexpanded—intravascular imaging lessons on coronary stent thrombosis

2018

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Predictors of Early, Late, and Very Late Stent Thrombosis After Primary Percutaneous Coronary Intervention With Bare-Metal and Drug-Eluting Stents for ST-Segment Elevation Myocardial Infarction

Abstract: Stent thrombosis after primary percutaneous coronary intervention is relatively frequent and continues to increase out to 5 years. New strategies are needed to prevent ST in STEMI patients, and targeted therapies are needed in patients identified at highest risk.

Cited by 61 publications

References 23 publications

Relation between stent thrombosis and calcium channel blocker after drug-eluting stent implantation

Relation between stent thrombosis and calcium channel blocker after drug-eluting stent implantation

Incidence and Clinical Predictors of Stent Restenosis and Early Stent Occlusion in Patients with Acute Myocardial Infarction Treated by Bare Metal Stents: Importance of Infarct Location and Serum Creatinine Level

Malapposed, uncovered, underexpanded—intravascular imaging lessons on coronary stent thrombosis

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