“…Neurologic manifestations of Lyme disease include meningitis, facial nerve palsy, cranial neuritis, motor or sensory radiculoneuritis, or subtle encephalitis [1]. Disease may be due, in part, to presence of the causative spirochete, Borrelia burgdorferi , within the nervous system, the virulence of particular organisms, and the host immune response to the pathogen [2–5]. Antibodies to the B. burgdorferi flagellin, elicited during the course of infection, have been shown to bind to human axons and therefore may be implicated in the pathogenesis of neurologic disease [1, 2, 6].…”