Preface

Bogaert, Ludo van; Radermecker, J

doi:10.1016/b978-1-4831-6677-3.50005-2

Cited by 3 publications

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“…Das Syndrom der Ophthalmoplegie mit Ataxie und Areflexie, das bereits 1932 von Collier (1) und 1938 von van Bogaert (14) erwähnt wurde, beschrieb Fisher (3) 1956 aus-fUhrlicher. Es stellt ein besonderes, einheitliches klinisches Krankheitsbild dar, das als Sonderform der idiopathischen Polyradiculoneuropathie Gzillain-Ba"e angesehen wird.…”

Section: Zusammenfassungunclassified

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Fisher-Syndrom (Ophthalmoplegie, Ataxie, Areflexie) bei einem 14jährigen Knaben

Bosch¹,

Loth

Fichsel³

1981

Klin Padiatr

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The syndrome of ophthalmoplegia, ataxia and areflexia first mentioned in 1932 by Collier and in 1938 by van Bogaert was described more precisely in 1956 by Fisher. It represents a specific clinical entity and is considered to be a variant of idiopathic polyradiculoneuropathy (Guillain-Barré). The most striking features are external (sometimes also internal) ophthalmoplegia, hyporeflexia or areflexia and cerebellar ataxia. During the first days paresthesias are frequently present whereas other disturbances of sensibility are not common. Additional signs of neurological lesions may be observed in association with the syndrome. Spinal fluid shows the typical albumino-cytological dissociation. The disease is characterized by its benign course. The etiology remains unclear, therapy is not necessary.

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“…The etiology remains unc1ear, therapy is not necessary. (14), die erst später die Bezeichnung ..Fisher-Syndrom" erhielt (3).…”

Section: Syndrome Of Ophthalmoplegia Ataxia and Areßexia Of Fisher Iunclassified

Fisher-Syndrom (Ophthalmoplegie, Ataxie, Areflexie) bei einem 14jährigen Knaben

Bosch¹,

Loth

Fichsel³

1981

Klin Padiatr

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“…Similar experience has been reported from other countries (Rawls, Dyck, Klass, Greer, and Herrmann, 1966;May, Dahn, and Reuss, 1967). This disease has also been described as acute necrotizing encephalitis (Van Bogaert, Radermecker, and Devos, 1955). It is characterized by widespread inflammation of the meninges and brain associated with severe necrosis and usually presents clinically as an acute pyrexial illness of the central nervous system.…”

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confidence: 99%

Electron microscopic observations on the virus particles inHerpes simplexencephalitis

Roy

Wolman

1969

J Clin Pathol

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Cerebral cortex from eight fatal cases ofHerpes simplexencephalitis has been studied by electron microscopy. The diagnosis had been based on clinical findings, necropsy evidence of bitemporal necrosis, and histological confirmation of intranuclear inclusions with a lymphocytic inflammatory response, even though virus cultures were unsuccessful. Virus particles were seen predominantly in nuclei in six of these cases, utilizing tissue which had been preserved in formalin for up to four years or had been embedded in paraffin. Although demonstration of these particles is only indicative of the herpes group and not specifically ofHerpes simplex, electron microscopy of either biopsy or postmortem material may be a useful adjunct in making the diagnosis and can be accomplished before virological studies have been completed.

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“…(4) our special requirements, but the New Appliances principles governing their design could be modified to suit any hospital. It is intended that at the hospital patients should be screened by an experienced surgeon and classified into: urgent cases requiring resuscitation in the specially prepared resuscitation area; major cases requiring operation or hospitalization or both, and who are sent to specially cleared wards; minor cases requiring outpatient treatment and subsequently to be sent home; and the dead,…”

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Documentation for a major incident.

Elson¹,

Eastwood²

1969

BMJ

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syndrome is a small testis, which is the only unequivocal sign always found. Whether this has got anything to do with his liability to all these other disorders I can't say. A lot of neurological and psychological disorders do occur with Klinefelter's syndrome.Professor BOOTH: Finally, I would very much like to thank Professor Henry Miller for coming and exposing himself to this exercise. It is always a problem-the relation between reading a piece of paper and what you see in a patient, and I think for us the tremendous value is to hear a very distintinguished and excellent clinician discussing a problem of this sort. It never matters whether you are right or wrong-what matters is to hear your mind thinking aloud. This has been a tremendous occasion for us, and quite the best Wednesday morning for me for a very long time. We are most grateful to you.We are grateful to Professor J. P. Shillingford and Dr. E. D. Williams for assistance in preparing this report, and to Mr. W. Brackenbury for the photomicrographs.The appointments held by the speakers at the time of the conference are listed below.

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Preface

Cited by 3 publications

References 0 publications

Fisher-Syndrom (Ophthalmoplegie, Ataxie, Areflexie) bei einem 14jährigen Knaben

Fisher-Syndrom (Ophthalmoplegie, Ataxie, Areflexie) bei einem 14jährigen Knaben

Electron microscopic observations on the virus particles inHerpes simplexencephalitis

Documentation for a major incident.

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