Preface

Newman, Michael G.; Takei, Henry H.; Klokkevold, Perry R.; Carranza, Fermín A.

doi:10.1016/b978-1-4377-0416-7.00109-8

Cited by 315 publications

(562 citation statements)

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“…Among these the biochemical parameters especially the enzymatic concentrations in the gingival crevicular fluid (GCF) pose as a potential tool to assess disease activity. Interestingly the most common enzymes found in the GCF are released from the poly morhonuclear leucocytes (PMN) and these cells play a pivotal role in the pathogenesis of periodontitis [9] and their numbers vary from patient to patient as well as from site to site.…”

Section: Introductionmentioning

confidence: 99%

Leucocyte Count: A Window to Periodontal Disease? A Pilot Study

Nair¹,

Ebenezer²,

Jalaluddin³

et al. 2014

IOSRJDMS

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Section: Introductionmentioning

confidence: 99%

Leucocyte Count: A Window to Periodontal Disease? A Pilot Study

Nair¹,

Ebenezer²,

Jalaluddin³

et al. 2014

IOSRJDMS

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“…Many forms of gingival and periodontal diseases have affected humans since the beginning 1 . Classification of periodontal diseases has changed over recent decades and, though a consensus between various global partnerships has been sought, agreement has not happened.…”

Section: Introductionmentioning

confidence: 99%

“…Others define it as the apical migration of the gingival margin along the root surface 1, 2, 5 . Carranza said recession is the exposure of the root surface by an apical shift in the position of the gum 1 . There are many surgical and nonsurgical procedures involving preventive and corrective methods for dealing with this diagnosis 6 .…”

Section: Introductionmentioning

confidence: 99%

Porcine collagen matrix for treating gingival recession. Randomized clinical trial.

Castro¹,

Grados²

2013

Joralres

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Achieving root coverage after exposure caused by gingival recession is one of the main goals of reconstructive periodontal surgery. Even though a large variety of techniques and mucogingival grafting procedures are available, their long-term results are not clear yet. Therefore, this study aimed to compare clinical effectiveness of the porcine collagen matrix with subepithelial connective graft for treating Miller class I and II gingival recessions. Materials and methods: The randomized clinical trial included twelve patients assigned to two groups. In the first group (experimental), six patients were treated using collagen matrix (mean age, 54.3 ± 5.6 years; mean recession 2. 67 ± 1.03 mm). Another group (control) of six patients was treated using connective grafts (mean age, 57.1 ± 2.7 years; mean recession 4.33 ± 1.03 mm). All patients underwent periodontal evaluation and pre-surgical preparation including oral hygiene instruction and supragingival scaling. Gingival recessions were exposed through partial thickness flaps where the grafts and matrices were placed. Patients were assessed periodically until complete healing of tissue. Results: Root coverage parameters, amount of keratinized gingiva, gingival biotype and clinical attachment level were evaluated. The root coverage percentage for the group using connective graft was 24.7 ± 13.5 % and 16.6 ± 26.8 % for the one treated with the matrix. The amount of increased keratinized tissue was 4.33 ± 2.06 mm and 4.5 ± 0.83 mm for the control and experimental group respectively. Both groups increased gingival biotypes from thin to thick at 100%. The final clinical attachment level was 4.17 ± 3.17 ± 04mm for the control group and 0.98 mm for the experimental group. There were significant differences between the outcome of gingival recession and clinical attachment. Conclusion: Results indicate both techniques, besides being predictable, are useful for improving clinical parameters when treating gingival recessions. Regardless of the fact that better root coverage was obtained with the subepithelial connective graft, both methods are really useful for increasing the thickness of keratinized tissue. Keywords: Gingival recession, connective tissue, grafts, periodontal disease, collagen matrix.Matriz de colágeno de origen porcino en el tratamiento de las recesiones gingivales. Ensayo clínico aleatorio.Resumen: Lograr la cobertura radicular que se expone por las recesiones gingivales es uno de los principales objetivos de las cirugías periodontales reconstructivas. Una gran variedad de técnicas e injertos mucogingivales se encuentran como opciones terapéuticas. Sin embargo, los resultados y efectividad a largo plazo aún no son claros. Este estudio tuvo como objetivo comparar la efectividad clínica de la matriz de colágeno de origen porcino versus el injerto conectivo subepitelial en el tratamiento de las recesiones gingivales Miller clase I y II. Materiales y métodos: Ensayo clínico aleatorizado donde participaron 12 pacientes asignados de manera aleatoria en...

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“…The genetic component does not cause the disease; however it does make patients more susceptible to emergence of periodontal pathologies or to more serious disease course. 44 Certain pathogens, such as Porphyromonas gingivalis, Tanerella forsythia and Fusobacterium nucleatum, can also influence disease severity. 3 Stress also contributes to increasing the incidence of PD by creating conditions of resistance to glucocorticoids and increased IL-1, IL-6 and TNF-α production, 45 caused by a dysfunction of CD11b monocytes in response to microbial products.…”

Section: Pathophysiology Of Periodontal Diseasementioning

confidence: 99%

Periodontal disease and atherosclerosis

et al. 2014

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Atherosclerotic disease (AD) is one of the most important causes of morbidity and mortality in the world. It expresses inflammatory markers such as C-reactive protein (CRP) and can provoke arterial wall thickening, which can be evaluated using Doppler ultrasound. Risk factors associated with AD include diabetes mellitus, systemic arterial hypertension, dyslipidemia and smoking. More recently, periodontal disease (PD) has been identified as a factor related to AD. Periodontal disease has a high prevalence in the global population and the inflammatory process and bacterial activity at the periodontium appear to increase the risk of AD. Encouraging good oral hygiene can reduce expression of inflammatory markers of AD. A review of literature on PD, AD and inflammatory markers and the interrelationships between the two diseases was conducted using data published in articles indexed on the PUBMED, SCIELO and BIREME databases.Keywords: periodontal disease; atherosclerosis; carotid intima-media thickness. ResumoA doença aterosclerótica (DA) constitui uma das principais causas de morbimortalidade no mundo. A sua expressão laboral pode ser através de marcadores inflamatórios, como a proteína C reativa (PCR) e/ou o espessamento da parede arterial, que pode ser analisado pela ultrassonografia com Doppler colorido. Os fatores de risco associados à DA são o diabetes mellitus, a hipertensão arterial sistêmica, a dislipidemia e o tabagismo. Mais recentemente, a doença periodontal (DP), que tem uma elevada prevalência na população mundial, tem sido considerada um fator relacionado ao desenvolvimento da DA, em que o processo inflamatório e a atividade bacteriana no periodonto parecem aumentar o risco para a DA. A motivação da higiene oral pode diminuir a expressão dos marcadores inflamatórios da DA. Com base em dados publicados em revistas eletrônicas e indexados pelos mecanismos de busca PUBMED, SCIELO e BIREME, foi realizada uma revisão de literatura sobre a DP e a DA, além dos marcadores inflamatórios expressos em ambas as doenças e suas possíveis inter-relações.

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Preface

Cited by 315 publications

References 0 publications

Leucocyte Count: A Window to Periodontal Disease? A Pilot Study

Leucocyte Count: A Window to Periodontal Disease? A Pilot Study

Porcine collagen matrix for treating gingival recession. Randomized clinical trial.

Periodontal disease and atherosclerosis

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