Pressure distribution at the ankle joint

Bruns, J.; Rosenbach, B.

doi:10.1016/0268-0033(90)90018-2

Cited by 37 publications

(23 citation statements)

References 27 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Extensive in vitro investigations have reported on ankle articular cartilage biomechanics using cadaveric experimental setup. [3][4][5][6][7][8][9][10][11][12][13][14][15][16] Carbon black transference techniques have been used to determine the contact area in the tibiotalar articulations. 3 Pressure sensors or pressure-sensitive film have been used to determine the contact areas and surface pressure distributions on the trochlea tali.…”

mentioning

confidence: 99%

“…3 Pressure sensors or pressure-sensitive film have been used to determine the contact areas and surface pressure distributions on the trochlea tali. [4][5][6][7][8][9] Kinematic tracking methods have also been used to determine ankle joint contact areas, 14,15 such as radiostereometric analysis (RSA) combined with three-dimensinal (3D) surface modeling method 14 or magnetic tracking device combined with proximity calculations of digitized joint surfaces. 15 However, no study has investigated the in vivo articular cartilage contact biomechanics in the ankle joint.…”

mentioning

confidence: 99%

See 1 more Smart Citation

In vivo cartilage contact deformation of human ankle joints under full body weight

Wan

Rubash

et al. 2008

Journal Orthopaedic Research

View full text Add to dashboard Cite

Quantitative data on in vivo deformation of articular cartilage is important for understanding the articular joint function and the etiology of degenerative joint diseases such as osteoarthritis. This study experimentally determined the in vivo cartilage thickness distribution and articular cartilage contact strain distribution in human ankle joints under full body weight loading conditions using a combined dual fluoroscopic and magnetic resonance imaging technique. The average cartilage thickness with the joint non-weight bearing was found to be 1.43 mm AE 0.15 mm and 1.42 mm AE 0.18 mm in the distal tibial and proximal talar cartilage layers, respectively. During weight bearing on a single leg, the strain distribution data revealed that 42.4% AE 15.7% of the contact area had contact strain higher than 15% in the ankle joint. Peak cartilage contact strain reached 34.5% AE 7.3%. This quantitative data on in vivo human cartilage morphology and deformation demonstrated that the cartilage may undergo large deformations under the loading conditions experienced in human ankle joints during daily activities. The in vivo cartilage contact deformation can be used as displacement boundary conditions in three-dimensional (3D) finite element models of the joint to calculate in vivo 3D articular cartilage contact stress/strain distributions. ß

show abstract

mentioning

confidence: 99%

mentioning

confidence: 99%

In vivo cartilage contact deformation of human ankle joints under full body weight

Wan

Rubash

et al. 2008

Journal Orthopaedic Research

View full text Add to dashboard Cite

show abstract

“…Das Supinationstrauma wird nicht nur für die lateralen sondern auch für die medialen osteochondralen Läsionen verantwortlich gemacht (17). Der Varusstress im OSG kann zu einer Impaktierung der medialen Talusrolle in die tibiale Gelenkfläche führen (Abb.…”

Section: N Traumatische Osteochondrale Läsionen Des Talusunclassified

Osteochondrale Läsionen des Talus

Steinhagen

Bruns

2003

Fuß & Sprunggelenk

Self Cite

View full text Add to dashboard Cite

“…Die Ätiologie der OCL des Talus ist noch immer nicht abschließend geklärt und prospektive Studien zur Evaluation der Ätiologie mit großen Patientenkollektiven existieren nicht [4,6,7]. Sowohl eine lokale Ischämie mit folgender Nekrose, avaskuläre Nekrosen, endokrine und metabolische Faktoren, als auch Achsfehlstellungen der Gelenke sind beschrieben worden [4,[8][9][10][11]. In der aktuellen Literatur konnte gezeigt werden, dass Varusoder Valgusfehlstellungen des Rückfußes zu einer Lastzunahme in dem entsprechenden Gelenkkompartiment des OSG führen [8,9].…”

unclassified

“…Sowohl eine lokale Ischämie mit folgender Nekrose, avaskuläre Nekrosen, endokrine und metabolische Faktoren, als auch Achsfehlstellungen der Gelenke sind beschrieben worden [4,[8][9][10][11]. In der aktuellen Literatur konnte gezeigt werden, dass Varusoder Valgusfehlstellungen des Rückfußes zu einer Lastzunahme in dem entsprechenden Gelenkkompartiment des OSG führen [8,9]. Zur radiologischen Bestimmung der Rückfußachse (RFA) hat sich die Rückfußaufnahme nach Saltzman bewährt [12][13][14][15].…”

unclassified