Prevalence and clinical presentation of cirrhotic cardiomyopathy: A single centre experience from southern India

A Prospective Study of Prevalence and Predictors of Cirrhotic Cardiomyopathy and Its Role in Development of Hepatorenal Syndrome

“…(Table4)J o u r n a l P r e -p r o o fThe prevalence of CCM in our study was 48% and most of the patients (84%) had a mild diastolic dysfunction. These results are almost similar to earlier reported studies (9)(18)(19). While mild diastolic dysfunction is common especially in elderly; more severe forms of diastolic dysfunction (Grade 2/3) are not frequently encountered.…”

The prevalence of cirrhotic cardiomyopathy according to different diagnostic criteria

“…Nevertheless, the agreement between the old and new recommendations was poor (k = −0.16, reclassification rate = 55.7%). Overall prevalence differs significantly rather because of different diagnostic criteria used in these studies [20][21][22] than population characteristics or disease frequency changes over time.…”

Evaluation of QTC Interval in Cirrhosis Patients With Variceal Hemorrhage

“…The QTc interval was prolonged in 75.8% of patients diagnosed with CCM. The risk factors like diabetes, alcohol, hypertension, hypothyroidism, which prolong the duration of QTc, were excluded in this study (2). In his study conducted on Hepatitis C (HCV) patients, Abbas et al found that QTc was longer than normal in 24.5% of these patients.…”

“…The prolongation in the QT interval causes increased heart rate, decreased myocardial contraction strength, and diastolic dysfunction. The deterioration in the transmission in the ventricle increases the risk of ventricular arrhythmia, especially polymorphic ventricular tachycardia, which can result in ventricular fibrillation and sudden death (2,4,5). It was shown in previous studies that the prolonged QT interval because of cirrhosis was directly proportional to the severity of cirrhosis, and there is healing after liver transplantation.…”

“…Cirrhosis causes cardiomyopathy (CCM), which causes weak contraction and relaxation of the heart. CCM has significant electrophysiological changes, which can be detected with Electrocardiogram (ECG) (1,2). Depolarisation and repolarisation of the ventricles are delayed because of the slow-down of the transmission in the myocardia, which, in return, causes the prolongation of QT interval in the ECG (3).…”