Prevalence of cerebral palsy in Slovenia: birth years 1981 to 1990

Kavčič, Anamarija; Perat, Milivoj Velickovic

doi:10.1111/j.1469-8749.1998.tb15396.x

Cited by 28 publications

(3 citation statements)

References 19 publications

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“…Whereas, in European countries spastic diplegia is seen in significantly higher rates. Studies reported the ratio of spastic quadriparesic CP as 18% -20.8% and spastic diplegic CP as 40.9% -54.9% in Europen countries [12] [16] [17]. The probable explanations of this finding may be the decreased perinatal mortality of premature babies as a result of more available and equipped newborn intensive care units in western countries and observed differences in predisposing risk factors in-between.…”

Section: Discussionmentioning

confidence: 99%

Epidemiologic Characteristics of Patients with Cerebral Palsy

Fidan¹,

Baysal²

2014

OJTR

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Aim: The aim of this study was to determine the demographical and clinical features of patients with cerebral palsy (CP) attending to our outpatient or inpatient clinics. Materials and Methods: Children admitted to Rehabilitation outpatient or inpatient clinics, Malatya, Turkey with the diagnosis of CP during the study period were included in the study. Age, sex, etiological factors, clinical classifications, and epidemiological characteristics such as socio-economic status as well as the problems associated with CP were analyzed in all patients. Results: Of the total of 130 patients with mean ages 51.05 ± 36.06 months, 51 were girls and 79 were boys. The most common etiologic risk factors were bleeding and presence of threatened miscarriage in prenatal period; asphyxia, low birth weight and prematurity in perinatal period; and neonatal convulsions and hyperbiluribinemia in the postnatal period. Consanguineous marriage was present in 24.6% of families. In clinical classification it was seen that 45 subjects were spastic quadriplegic (34.6%), 41 were spastic diplegic (31.5%), 15 were dyskynetic (11.5%), 14 were spastic hemiplegic (10.8%), 10 were mixed type (7.7%) and 5 were hypotonic/ataxic (3.8%). Conclusion: Prenatal risk factors constitute the foremost risk factors in CP etiology. CP incidence can be lowered by close follow up of the newborns who has been considered as high risk newborns due to the presence of risk factors during pregnancies or deliveries, and by increasing the number of neonatal intensive care units. Besides, it is important to raise public awareness about consanguinity marriages in our country.

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Section: Discussionmentioning

confidence: 99%

Epidemiologic Characteristics of Patients with Cerebral Palsy

Fidan¹,

Baysal²

2014

OJTR

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“…Escobar (1989) acredita que na Espanha a incidência seja de 1,5 a 6 por 1000 dos indivíduos da população com menos de 21 anos de idade. Na Eslovênia, a prevalência tem mantido-se constante em crianças nascidas com mais de 38 semanas de gestação e 2500 gramas de peso ao nascimento, sendo as síndromes espásticas as mais encontradas, acometendo 84.8% das crianças (Kavcic, 1998). Entretanto, este autor encontrou uma diminuição da incidência de 3.3 por 1.000 nascimentos em 1981 para 2.3 por 1.000 em 1990 nas crianças nascidas com até 38 semanas de gestação e 1500 gramas de peso ao nascimento.…”

Section: I1 Conceitosunclassified

“…A PC do tipo espástica é encontrada com maior freqüência tanto nos bebês a termo como pré-termo independente de sua etiologia (Escobar et al, 1989;Tabuse, 1992;Schenk-Rootlieb et al, 1994;Kavcic, 1998). Entretanto, com o aumento da sobrevivência de bebês com baixa idade gestacional com peso muito baixo proporcionados pelos recentes avanços tecnológicos, a proporção de todos os grupos de PC devida a complicações perinatais está sofrendo alterações, assim como a severidade dos quadros (Hagberg et al, 1989).…”

Section: Mistounclassified

Acuidade visual de resolução de grades em crianças com paralisia cerebral do tipo espástico pelo método dos potenciais visuais evocados de varredura

Costa¹

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Dissertação defendida e aprovada em: ___/ ___/ ___Dedico este trabalho aos meus pais, Manoel e Rosa, ao meu irmão Rodrigo pelo constante incentivo, apoio, amor e carinho. À Rita por tão carinhosamente compreender os meus momentos de ausência ao seu lado em prol da pesquisa. Às crianças da AACD todo o meu respeito, amor e admiração. "Bem aventurados os que choram e sofrem, porque serão consolados"

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Cerebral palsy in very preterm infants: new epidemiological insights

O’Shea

2002

Ment. Retard. Dev. Disabil. Res. Rev.

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The focus of this review is on new insights from recent epidemiological research on cerebral palsy in preterm infants. These include: 1) a better understanding of issues related to diagnosis and classification; 2) new information about the brain abnormalities underlying cerebral palsy in preterm infants; and 3) a better understanding of biological mechanisms that may underlie previously described epidemiological associations. Ongoing efforts to improve the diagnosis and classification of cerebral palsy have been enhanced by findings from serial examinations of cohorts of very preterm infants. Cranial ultrasonography through the anterior fontanelle of very preterm infants has provided information about grossly evident brain damage, found in about one-half of preterm infants who develop cerebral palsy. Insights into the pathophysiologic basis for certain epidemiologic associations have come from studies of experimental brain damage in animals and clinical studies of neurologic disorders in adults. Much of the current epidemiological research into the causes of cerebral palsy in preterm infants has focused on two potential mechanisms of brain damage. One mechanism involves insufficient cerebral perfusion; the other, cytokine-mediated damage, potentially triggered by events such as maternal infection (e.g., intrauterine or periodontal infection), neonatal infection (e.g., sepsis and necrotizing enterocolitis), and neonatal oxygen- or ventilator-induced lung injury. In addition to the preterm infant's increased exposure to such damaging factors, the high frequency of cerebral palsy in these infants might be due, in part, to insufficient levels of developmentally regulated protective substances, such as thyroid hormone and glucocorticoids. Models of causation currently are being investigated using recently developed methods for quantifying, with small quantities of blood, biomolecules that are suspected to either promote or protect against brain damage in the neonate. Clinical investigations now under way can be expected to identify strategies to be tested in clinical trials that could lower the risk of cerebral palsy in very preterm infants.

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Prevalence of cerebral palsy in Slovenia: birth years 1981 to 1990

Cited by 28 publications

References 19 publications

Epidemiologic Characteristics of Patients with Cerebral Palsy

Epidemiologic Characteristics of Patients with Cerebral Palsy

Acuidade visual de resolução de grades em crianças com paralisia cerebral do tipo espástico pelo método dos potenciais visuais evocados de varredura

Cerebral palsy in very preterm infants: new epidemiological insights

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