Prevalence of Pericardial Effusions in Children With Large Atrial or Ventricular Septal Defect

Reddy, Surendranath; Reddy, Shiva; Thomas, Ronald; Ross, Robert D.

doi:10.1016/j.amjcard.2008.08.065

Cited by 4 publications

(2 citation statements)

References 7 publications

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“…As previously mentioned, congenital atrial septal defects increase the risk of developing a pericardial effusion. In a retrospective study, analyzing the prevalence of pericardial effusions in children with ASD compared to those with ventricular septal defect (VSD) and those with normal cardiac anatomy, 32% of children (n=81) with a known ASD also had an effusion present in the pericardial space [ 8 ]. In addition, it is known that infections of the myocardium may induce an inflammatory response, leading to the accumulation of fluid in the pericardium.…”

Section: Discussionmentioning

confidence: 99%

Congenital Atrial Septal Defect Presenting With Tamponade Physiology and an Associated Viral Illness

Juarez,

Jhon,

Khouzam

et al. 2023

Cureus

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Cardiac tamponade is a serious clinical syndrome that often presents with the classic triad of hypotension, jugular vein distention and diminished or muffled heart sounds on auscultation (Beck’s Triad). This phenomenon occurs due to fluid accumulation in the pericardial space which compresses the heart, reduces cardiac output and may cause cardiogenic shock. In this report, we present a case of a 22-year-old female with a congenital atrial septal defect (ASD) and right ventricular failure with tamponade physiology with an associated viral illness.

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Section: Discussionmentioning

confidence: 99%

Congenital Atrial Septal Defect Presenting With Tamponade Physiology and an Associated Viral Illness

Juarez,

Jhon,

Khouzam

et al. 2023

Cureus

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“…The second patient had mild to moderate pre-existing PE with no response to anti-inflammatory agents during 2- to 3-month follow-up that seems to be due to ASD itself. With mild to moderate pre-existing PE, Reddy et al speculated that, if septal defects were left untreated, the effusion may accumulate over time because of chronic volume overload, which may lead to more PE [ 9 ]. The amount of PE in this patient also increased significantly after two ASD device implantations that necessitate pericardiocentesis.…”

Section: Discussionmentioning

confidence: 99%

Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous?

et al. 2021

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Background Secundum-type atrial septal defects (ASD) constitute 8% to 10% of congenital heart defect. Secundum ASDs can be closed either percutaneously or surgically. However, ASD device closure has proven to be technically safe and feasible; it is not free of complications. These complications include device embolization/malposition which have been reported in 3.5% of cases, arrhythmia, and pericardial effusion in 2.6% and 0.5–1.5% respectively, device thrombus, residual shunting, and impingement of the device on the adjacent structures. Case presentation We introduce three patients with secundum ASD who had preexisting pericardial effusion, device closure was performed for them, and after the procedure, the effusion size progressed significantly. We used multimodality imaging to diagnose the cause of pericardial effusion (PE). Cardiac erosion was diagnosed in one of the patients that managed surgically. We did not found any specific procedure-related cause for worsening the pericardial effusion in the other two patients. Conclusion Several reasons include procedure-related complication and other systemic causes should be considered in patients who develop pericardial effusion after trans catheter closure of ASDs.

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Early Repair of Large Infant Ventricular Septal Defect Despite Respiratory Syncytial Virus–Induced Respiratory Failure With Postrepair Chylous Pericardial Effusion Requiring Pleuropericardial Window

Sykes¹,

Verma²,

Peshkovsky³

et al. 2012

Pediatric Emergency Care

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The surgical correction of congenital cardiac lesions that are complicated by intercurrent respiratory syncytial virus (RSV) pneumonitis has traditionally been deferred for at least 6 to 8 weeks. The presumption is that using cardiopulmonary bypass will increase the risk of postoperative pulmonary complications. We present an infant who developed acute respiratory failure related to RSV pneumonitis and required urgent mechanical ventilation. Cardiac evaluation revealed a large nonrestrictive ventricular septal defect (VSD), aortic arch hypoplasia, normally functioning bicuspid aortic valve, and hemodynamic instability associated with markedly increased pulmonary blood flow. Separation from mechanical ventilation was unsuccessful preoperatively. He underwent VSD repair with cardiopulmonary bypass less than 4 weeks after initial RSV infection. He was extubated successfully within 72 hours of VSD repair. Approximately 6 weeks postoperatively, he developed a circumferential chylous pericardial effusion of unclear etiology--an exceedingly rare complication of VSD repair in early infancy in a non-Down syndrome patient. The chylous effusion was initially managed unsuccessfully with Portogen/Monogen and a percutaneously placed pericardial drain. Two weeks later, he underwent creation of a pleuropericardial window with successful resolution of the chylous effusion. It is of interest to pediatricians to be able to correctly time the repair of congenital heart disease lesions after RSV infection to minimize post-bypass pulmonary complications and yet avoid morbidity from undue delays in repair. In addition, chylopericardium can occur in infants after VSD repair, and dietary modification and catheter drainage may not be adequate.

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Prevalence of Pericardial Effusions in Children With Large Atrial or Ventricular Septal Defect

Cited by 4 publications

References 7 publications

Congenital Atrial Septal Defect Presenting With Tamponade Physiology and an Associated Viral Illness

Congenital Atrial Septal Defect Presenting With Tamponade Physiology and an Associated Viral Illness

Case report of worsening of preexisting pericardial effusion after ASD device closure: is it calamitous?

Early Repair of Large Infant Ventricular Septal Defect Despite Respiratory Syncytial Virus–Induced Respiratory Failure With Postrepair Chylous Pericardial Effusion Requiring Pleuropericardial Window

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