Protective effects of cinnamic acid and cinnamic aldehyde on isoproterenol-induced acute myocardial ischemia in rats

Salidroside Mitigates Sepsis-Induced Myocarditis in Rats by Regulating IGF-1/PI3K/Akt/GSK-3β Signaling

“…Exogenously supplied IGF-1 has also been proven to improve left ventricular function and reduce cardiomyocyte apoptosis after experimental coronary artery ligation in Sprague Dawley rats [23]. Sal can relieve myocardial inflammation cell infiltration, inhibit cardiomyocyte apoptosis, and reduce myocardial ischemia-reperfusion injury [24,25]. Furthermore, it has been reported that growth factors could improve cell survival by activating PI3K and its downstream gene Akt in different cell types [26].…”

Antioxidation Role of Different Lateral Stellate Ganglion Block in Isoproterenol-Induced Acute Myocardial Ischemia in Rats

“…47 (3) Myocardial ischemia induced by AMI could increase NO expression after oxidative stress injury in rat heart and SG. 47,48 Tissue acidosis occurring during ischemia also could increase NO production. 49 FIGURE 4.…”

Screening of Polyphenol Composition and Antiradical Capacity of Wild Erica arborea: A Native Plant Species from the Portuguese Flora

“…They may exert protection of cell structures through different mechanisms, and one in particular is their ability to increase the levels of the powerful intracellular antioxidant enzymes [25,26]. Also, recent works have demonstrate that phenolic acids like chlorogenic and neochlorogenic acids can have a strong anti-inflammatory activity [27,28]. Similar observations were made recently to caffeic acid by Song et al [27] who reported that caffeic acid is able to inhibit the activity of acetylcholinesterase (AChE) and butyrylcholinesterase (BChE) in a dose-dependent manner, protecting the brain against neuronal inflammatory processes and diseases.…”