Public health importance of triggers of myocardial infarction: a comparative risk assessment

Nawrot, Tim S.; Pérez, Laura; Künzli, Nino; Munters, Elke; Nemery, Benoît

doi:10.1016/s0140-6736(10)62296-9

Cited by 474 publications

(300 citation statements)

References 72 publications

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“…The study designs to identify such ''agents'' require both frequent contact with participants and better methods of identification of possible ''agents,'' e.g. air pollution, psychosocial stressors, infections, and their relationship to ''vulnerable plaques'' and thrombosis [62][63][64][65]. If inflammation plays an important role in conversion from atherosclerosis to clinical disease, then epidemiology studies need better measures of ''inflammation,'' e.g.…”

Section: Discussionmentioning

confidence: 99%

The limitations of opportunistic epidemiology, pseudopod epidemiology

Kuller

2016

Eur J Epidemiol

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Epidemiology has been remarkably successful in the past in identifying the important agents of disease, the impact of the environment, both physical and social, and interrelationship with host susceptibility (genomics). Many of the advances in improving the health of individuals and populations have been the result of epidemiology studies that have identified the specific ''agents'' of disease and application of public health and preventive medicine. In recent years, large longitudinal studies have dominated epidemiology research, especially long incubation period chronic diseases. The initial hypotheses in these studies have been expanded by vertical extension studies using newer technologies to measure independent variables, vertical pseudopods, and additional studies of other diseases, horizontal pseudopods, of the original longitudinal study. Host susceptibility, i.e. genomics, has also become a prominent component of these longitudinal studies. The critical question addressed in this paper is whether these ''pseudopod'' epidemiology approaches have enhanced public health or generated a large number of studies of little impact.

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Section: Discussionmentioning

confidence: 99%

The limitations of opportunistic epidemiology, pseudopod epidemiology

Kuller

2016

Eur J Epidemiol

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“…outbursts of anger or grief). 96 After the death of a significant person, the incidence rate of acute myocardial infarction (AMI) is elevated 21-fold during the first 24 hours, declining steadily during the subsequent days. 97 Chronic stress at work (e.g.…”

Section: Epigeneticsmentioning

confidence: 99%

2016 European Guidelines on cardiovascular disease prevention in clinical practice

Piepoli¹,

Hoes²,

Agewall

et al. 2016

Revista Española de Cardiología (English Edition)

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“…See (Levy et al 2010) for details of established sideeffects of these pollutants. It is also important to note recent advances in health science where the link between heart attack triggers and urban pollution is hypothesized (Nawrot et al 2011). …”

Section: Introductionmentioning

confidence: 99%

Markov Chain Based Emissions Models: A Precursor for Green Control

Crisostomi

Kirkland

Schlote

et al. 2011

Green IT: Technologies and Applications

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In this chapter we propose a new method of modelling urban pollutants arising from transportation networks. The efficacy of the proposed approach is demonstrated by means of a number of examples. Our models give rise to a number of surprising observations that are relevant for the regulation of pollution in urban networks: Different actions are required for the control of different pollutants and low speed limits do not necessarily lead to low pollution.

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Public health importance of triggers of myocardial infarction: a comparative risk assessment

Cited by 474 publications

References 72 publications

The limitations of opportunistic epidemiology, pseudopod epidemiology

The limitations of opportunistic epidemiology, pseudopod epidemiology

2016 European Guidelines on cardiovascular disease prevention in clinical practice

Markov Chain Based Emissions Models: A Precursor for Green Control

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