Pulmonary oedema associated with airway obstruction

Lang, Scott A.; Duncan, Peter G.; Shephard, David A. E.; Ha, Hung C.

doi:10.1007/bf03005472

Cited by 260 publications

(179 citation statements)

References 72 publications

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“…6 In this case report, the disruption of the equilibrium originated from the intrathoracic negative pressure secondary to suctioning of the ETF adapter. Multi-factorial pathogenesis of NPPE has been described by Lang et al 6 Intra-thoracic negative pressure increases venous return to the right heart and decreases the output of the left ventricle. This results in an increase of pulmonary blood volume and micro-vascular pressure, followed by pooling of blood in the lungs, loss of capillary integrity, redistribution of blood volume to the pulmonary system, decrease in the fimctional residue volunle, hypoxia and myocardial depression.…”

Section: Discussionmentioning

confidence: 98%

“…6 Laryngospasm is one of the most common causes of post-anaesthetic upper airway obstruction. 6,7 In this case there was no evidence of upper airway obstruction such as laryngospasm, falling tongue, etc., following extubation, nor difficult ventilation during bag/mask resuscitation.…”

Section: Discussionmentioning

confidence: 99%

“…This results in an increase of pulmonary blood volume and micro-vascular pressure, followed by pooling of blood in the lungs, loss of capillary integrity, redistribution of blood volume to the pulmonary system, decrease in the fimctional residue volunle, hypoxia and myocardial depression. 6 Chest ganglionectomy is an effective treatment for hyperhidrosis. It is a very short procedure for a relatively common disease in this subtropical country.…”

Section: Discussionmentioning

confidence: 99%

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Negative pressure pulmonary oedema induced by direct suctioning of endotracheal tube adapter

et al. 1998

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Section: Discussionmentioning

confidence: 98%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Negative pressure pulmonary oedema induced by direct suctioning of endotracheal tube adapter

et al. 1998

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“…These are due to changes in Starling forces of pulmonary circulation, hemodynamic changes secondary to negative intrathoracic pressure, as well as hypoxia and a hyperadrenergic state. 4 Tracheostomy or leaving the tube in situ for a few days are well recognized methods for managing tracheomalacia. 5 Had extubation been unsuccessful we would have proceeded to carry out a tracheostomy as distal as possible to enable location below the involved tracheal segment.…”

Section: Negative Pressure Pulmonary Edema Following Thyroidectomymentioning

confidence: 99%

Negative pressure pulmonary edema following thyroidectomy

Sharma¹,

Beckett²,

Gormley³

2002

Can J Anesth/J Can Anesth

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“…2,3 We report a case where a patient undergoing carotid artery stenting as a neuroradiological procedure developed sudden pulmonary edema, the cause of which became apparent only after retrospective review of clinical data.…”

mentioning

confidence: 99%

Pulmonary edema in the neuroradiology suite: a diagnostic dilemma

Quader

Manninen

Lai

2001

Can J Anesth/J Can Anesth

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CARDIOTHORACIC ANESTHESIA, RESPIRATION AND AIRWAYPurpose: To present the case of an initially unexplained complication of sudden pulmonary edema in a patient during stenting of the carotid artery in the interventional neurology suite.Clinical Features: A 46-yr-old woman (ASA III) having an intracavernous carotid artery angioplasty and stenting procedure under neurolept anesthesia developed sudden pulmonary edema on completion of an otherwise uneventful stenting procedure. The patient responded well to initial management of pulmonary edema although the cause of the edema remained unclear. On retrospective examination of data and with the evolution of clinical signs it became apparent that the patient had suffered a subarachnoid hemorrhage which eventually resulted in her death.Conclusions: Clinical signs of intracerebral events may be slow to evolve. The cause of sudden pulmonary edema in patients undergoing an interventional neuroendovascular procedure is likely to be neurogenic in origin despite the initial lack of neurological signs. Éléments cliniques : Une femme de 46 ans (ASA III) admise pour une angioplastie du siphon carotidien et la pose d'un tuteur sous neuroleptanesthésie, a développé un oedème pulmonaire soudain à la fin de la pose du tuteur, par ailleurs sans incident. La patiente a bien réagi au traitement initial de l'oedème pulmonaire même si la cause en est restée obscure. À la suite d'un examen rétrospectif des données et de l'évolution des signes cliniques, il est devenu évident que la patiente a subi une hémorragie sous-arachnoïdienne qui a finalement causé sa mort. Conclusion :Les signes cliniques d'événements intracérébraux peuvent présenter une lente évolution. La cause d'un oedème pulmonaire soudain chez des patients qui subissent une intervention neuroendovasculaire peut être d'origine neurogène malgré l'absence initiale de signes neurologiques.

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Pulmonary oedema associated with airway obstruction

Cited by 260 publications

References 72 publications

Negative pressure pulmonary oedema induced by direct suctioning of endotracheal tube adapter

Negative pressure pulmonary oedema induced by direct suctioning of endotracheal tube adapter

Negative pressure pulmonary edema following thyroidectomy

Pulmonary edema in the neuroradiology suite: a diagnostic dilemma

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