“…Typical consequences include distended pleural-hilar bronchial veins, alveolar haemorrhage, a friable endobronchial mucosa, a reduced lymphatic drainage, interstitial pulmonary oedema, enlarged hilar lymph nodes, enlarged lymph vessels and sometimes pleural effusions [77][78][79][80][81][82]. To keep the lung an optimal gas-exchanging system, the pulmonary arterial blood flow is also affected, with redistribution of the pulmonary arterial blood flow towards regions with lower vascular resistance [83][84][85][86][87]. In severe stenosis even a reversal flow in the pulmonary arteries with development of pulmonary venous hypertension, pulmonary arterial remodelling and a decreased arborisation of the pulmonary arterial tree may develop [88,89] (table 2).…”